Unvaccinated children healthier

Actually, the number is 0.

I'm curious as to how you know this to be fact Jeff. One of the stats
I saw on TV the other day I found interesting - 1 in 97 boys will be
diagnosed with some form of autism and that the ages of diagnosis or
onset is from something like 18 months to 6 years. Isn't that
precisely when these children, or at least just after that first tough
year of the multitude of vaccines, receive all the majority of their
vaccines for school entry?

On 14 Apr 2007 22:10:03 -0700, "Chris" wrote:

I'm curious as to how you know this to be fact Jeff. One of the stats
I saw on TV the other day I found interesting - 1 in 97 boys will be
diagnosed with some form of autism and that the ages of diagnosis or
onset is from something like 18 months to 6 years. Isn't that
precisely when these children, or at least just after that first tough
year of the multitude of vaccines, receive all the majority of their
vaccines for school entry?

First of all, the incidence of autism has increased as the incidence
of mental retardation and other diagnoses has decreased. Part of the
increase, then is due to better understanding of autism and better
diagnoses.

Second, the incidence increased because the diagnostic standards have
changed to include PDD-NOS and Asperger's which were not diagnosable
until around 1990.

Third, most autism is *not* the regressive kind of autism. For the
children I know, most did not regress, they simply did not progress in
the way that *normal* children did. The disorder becomes more
noticeable at around 18 months because that is when most parents see
the differences from the NT children of their friends. Many first
time parents don't notice these differences at all until children hit
preschool and they see how many quirks their children have in
comparison to NT three year olds. My grandson was *in his own world*
probably from the very beginning of his life.

Fourth, the fact that there is a correlation between time of noticing
onset and time of vaccination does not mean that the vaccines were the
cause of the condition.


--
Dorothy

There is no sound, no cry in all the world
that can be heard unless someone listens ..

The Outer Limits

"Chris" wrote in message
ups.com...


Actually, the number is 0.

I'm curious as to how you know this to be fact Jeff. One of the stats
I saw on TV the other day I found interesting - 1 in 97 boys will be
diagnosed with some form of autism and that the ages of diagnosis or
onset is from something like 18 months to 6 years. Isn't that
precisely when these children, or at least just after that first tough
year of the multitude of vaccines, receive all the majority of their
vaccines for school entry?

yes, but if you compare the videos of kids before their first birthdays who
later develop autism to those who don't, you can see differences in the
behavior of the two sets kids, especially social behaviors (this is done in
a blind manner, meaning that the people who are watching the kids don't know
which ones will become autistics later).

All of the evidence indicates that vaccination does not cause autism.

Jeff

On Apr 12, 11:11 pm, (David Wright) wrote:
In article .com,



bigvince wrote:
On Apr 12, 2:09 pm, (David Wright) wrote:
In article ,

JOHN wrote:

"Robert" wrote in message
...

I don't know of anything that a herb or nutrient has cured.

Herbs cure most things, so do nutrients--vitamin c will cure all infections
for examplehttp://www.whale.to/a/levy_h.html
"I have had hundreds and hundreds of stroke cases. My methods cured

"We've had numerous people diagnosed with Alzheimer's who got better; the

There is no definitive diagnosis of Alzheimers other than autopsy.

-- David Wright :: alphabeta at prodigy.net
These are my opinions only, but they're almost always correct.
"HPV shots don't cause promiscuity. Tequila shots do." -- Bill Maher

So all those scrips for Alzheimer's meds are based on what.? A
hunch?

Pretty much. And those meds don't work very well, either. Marginal
improvement at best.

and all those studies showing improvement as a result of
treating a disorder that cannot be definitively diagnosed until the
patient dies are based on bogus science.

The studies don't show a lot of improvement, I'm afraid. My personal
guess is that there will be some way to prevent AD well before there
is a way to treat it.

-- David Wright :: alphabeta at prodigy.net
These are my opinions only, but they're almost always correct.
"HPV shots don't cause promiscuity. Tequila shots do." -- Bill Maher

Agreed. Vince

"Jeff" wrote in message
news:1ZuUh.643$0S1.553@trnddc01...

"Chris" wrote in message
ups.com...


Actually, the number is 0.

I'm curious as to how you know this to be fact Jeff. One of the stats
I saw on TV the other day I found interesting - 1 in 97 boys will be
diagnosed with some form of autism and that the ages of diagnosis or
onset is from something like 18 months to 6 years. Isn't that
precisely when these children, or at least just after that first tough
year of the multitude of vaccines, receive all the majority of their
vaccines for school entry?

yes, but if you compare the videos of kids before their first birthdays
who later develop autism to those who don't, you can see differences in
the behavior of the two sets kids, especially social behaviors (this is
done in a blind manner, meaning that the people who are watching the kids
don't know which ones will become autistics later).

All of the evidence indicates that vaccination does not cause autism.

Jeff
Repeated lie.

http://www.thecre.com/quality/2005/2...f_quality.html

Thursday, June 16, 2005
Why Won't the CDC Allow Access to the Vaccine Safety Datalink?

Memo to CDC: We're not getting our money's worth
David Kirby
May 23, 2005

Can mercury in vaccines cause autism in children? This hotly disputed
question will only burn brighter as more biological evidence surfaces to
suggest a link. But a definitive answer might take years. Meanwhile, the
Centers for Disease Control and Prevention is sitting on a
multi-million-dollar database - paid for by you and me - that could probably
resolve this contretemps within weeks.

They have the data. We paid for the data. Yet we cannot see the data. The
information is kept under lock and key within the massive health agency --
as jealously guarded as nuclear secrets.

The CDC tells us that they have looked at the data exhaustively and found
"no evidence of harm." They implied that their own scientists are perfectly
capable of analyzing the data, thank you very much, and outside researchers
cannot be trusted to independently verify their analyses, nor to protect the
confidentiality of patients whose numbers they would be crunching.

But, as any high school student can tell you, the replication of a study is
the hallmark of all good science. Without access to the raw data originally
used by the CDC researchers, it is impossible to verify their work. All we
can do is trust that they got it right.

The CDC, which has budgeted nearly $200 million to operate the Vaccine
Safety Datalink, spent four years analyzing data from children who received
varying amounts of thimerosal in their vaccines. The study went through five
different permutations before being published in November, 2003. Early study
"generations," which were never meant to see the light of day, showed highly
elevated, statistically significant increased risks for autism and other
disorders among the kids receiving the most mercury.

But by the time the study was published, most of these associations had
somehow disappeared entirely.

Only two outside researchers, Mark and David Geier, have managed to gain
access to the raw CDC data. They faced daunting hurdles to get into the CDC
computer center, and nearly crippling software malfunctions once they were
inside. But among the data they did manage to mine, they reportedly found
highly elevated risks for autism among children in the highest mercury
exposure group.

So we now have two extremely different interpretations of the same data. It
is way past time that the CDC allow a third team - outside researchers
completely acceptable to all parties involved in this dispute - into the
database to conduct any analyses they see fit. (Patients names are removed
from the data, making it exceedingly hard for researchers to identify
anyone, even if they desired, which is extremely unlikely in itself).

It sounds reasonable, it sounds nice. But don't hold your breath. The CDC is
hardly issuing engraved invitations to come trawl its mainframes, despite a
harshly written report earlier this year from the Institute of Medicine. The
IOM complained of CDC foot dragging, and even insolence, on this matter, and
suggested that vaccine officials at the health agency seek "legal counsel."
Why? Because the original datasets of children used by the government have,
as they say, gone missing. (Actually, the official explanation was that they
"were not archived in a standard fashion.") The intentional loss or
destruction of taxpayer funded data or datasets is a violation of the
Federal Data Quality Act. It is a felony, and someone could go to jail for
it.

Meanwhile, the data just sit there. Our data, not theirs. CDC officials
insist they have an "open mind" on this issue, and that thimerosal has not
been ruled out as a possible cause of autism and other disorders. But they
also insist that the vaccine safety database yielded no evidence of harm.

If that is true, then why are they so reluctant to let someone else in to
verify this claim? I cannot answer that question, because the CDC is not
talking to me. But I do know that people with nothing to hide are
unencumbered by doubts of what others will find if they rifle through their
closet.

If the data can prove that injecting a known neurotoxin into infants at
levels up to 125 times over federal safety limits was a safe and sane thing
to do, then why isn't the CDC having an open house for all researchers worth
their salt to come on down and have a look-see for themselves?

Without access to the raw data, parents who support the thimerosal theory -
and their allies in Congress, academia and law - are falling back on other
recent studies that show a possible link between mercury and autism. They
may not have the epidemiology on their side, yet, but the mounting evidence
emerging from the fields of biology and toxicology is becoming too urgent to
ignore. Recent published studies have shown:

+ Autistic children retain mercury at much higher rates than non-autistic
kids.

+ Autistic children lack certain sulfur-based proteins that bind to heavy
metals and remove them from the body.

+ Autistic children have a dysfunctional immune profile generally consistent
with mercury toxicity.

+ The rate of increase in reported autism cases peaked between 1987 and
1992, the same years that new mercury-containing vaccines were added to the
U.S. schedule.

+ Mice with autoimmune disorders react horrifically to mercury exposure from
vaccines, whereas typical mice of the same species do not.

+ In primates, mercury from vaccines was more likely to become trapped in
the brain than mercury from fish.

+ Children who live near mercury spewing power plants have an elevated risk
of developing autism.

These are all intriguing, to be sure. But what we really need is to get our
hands on the raw CDC data - our data.

David Kirby is author of "Evidence of Harm" (St. Martin's Press)
www.evidenceofharm.com

http://www.flu.org.cn/news/2004986362.htm
Thimerosal,New study reopens debate on vaccinations
Published: Sep ,8,2004 16:21 PM
By ###
Special to The Wall Street Journal & Medicalnewstoday



By Tara Parker-Pope
The Wall Street Journal

Just a few months after the nation's top medical adviser rejected a link
between vaccines and autism, a mouse study has reignited the debate and
raised new fears among parents considering vaccinations and flu shots for
their kids.


For years, a cadre of parents and physicians have contended that thimerosal,
an ethyl-mercury compound that has been one of the most widely used vaccine
preservatives, is partly responsible for an apparent rise in autism in
recent decades. But broad population studies haven't supported the claim. In
May, a major report from the Institute of Medicine's Immunization Safety
Review Committee rejected a link between autism and vaccines.



But today, a congressional committee will review a June study from Columbia
University, which found that a preservative used in vaccines can cause
autism-like symptoms in a specific strain of mice. The research raises
questions about whether some people might be genetically vulnerable to the
effects of thimerosal.



The study also raises questions about a new push by the Centers for Disease
Control and Prevention to add flu shots to the immunization schedule for
school-age kids. The vast majority of flu shots given still contain the
preservative.



In the study, researchers administered thimerosal to four strains of young
mice. Three of the mice strains were unaffected by thimerosal, but the
fourth developed problems consistent with autism such as delayed growth,
social withdrawal and brain abnormalities. The mice were known to have a
genetic susceptibility to mercury.



Thimerosal, found in childhood vaccines, can increase the risk of
autism-like damage in mice



A new study indicates that postnatal exposure to thimerosal, a mercury
preservative commonly used in a number of childhood vaccines, can lead to
the development of autism-like damage in autoimmune disease susceptible
mice. This animal model, the first to show that the administration of
low-dose ethylmercury can lead to behavioral and neurological changes in the
developing brain, reinforces previous studies showing that a genetic
predisposition affects risk in combination with certain environmental
triggers. The study was conducted by researchers at the Jerome L. and Dawn
Greene

Infectious Disease Laboratory at the Mailman School of Public Health,
Columbia University. Over the past 20 years, there has been a striking
increase--at least ten-fold since 1985--in the number of children diagnosed
with autism spectrum disorders. Genetic factors alone cannot account for
this rise in prevalence. Researchers at the Mailman School, led by Dr. Mady
Hornig, created an animal model to explore the relationship between
thimerosal (ethylmercury) and autism, hypothesizing that the combination of
genetic susceptibility and environmental exposure to mercury in childhood
vaccines may cause neurotoxicity.

Cumulative mercury burden through other sources, including in utero
exposures to mercury in fish or vaccines, may also lead to damage in
susceptible hosts. Timing and quantity of thimerosal dosing for the mouse
model were developed using the U.S. immunization schedule for children, with
doses calculated for mice based on 10th percentile weight of U.S. boys at
age two, four, six, and twelve months.

The researchers found the subset of autoimmune disease susceptible mice with
thimerosal exposure to express many important aspects of the behavioral and
neuropathologic features of autism spectrum disorders, including:

Abnormal response to novel environments;

Behavioral impoverishment (limited range of behaviors and decreased
exploration of environment); Significant abnormalities in brain
architecture, affecting areas subserving emotion and cognition; Increased
brain size.

These findings have relevance for identification of autism cases relating to
environmental factors; design of treatment strategies; and development of
rational immunization programs. The use of thimerosal in vaccines has been
reduced over the past few years, although it is still present in some
influenza vaccines. Identifying the connection between genetic
susceptibility and an environmental trigger for autism--in this case
thimerosal exposure--is important because it may promote discovery of
effective interventions for and limit exposure in a specific population,
stated the lead author Dr. Mady Hornig. Because the developing brain can be
exposed to toxins that are long gone by the time symptoms appear, clues
gathered in these animal models can then be evaluated through prospective
human birth cohorts--providing a powerful to tool to dissect the interaction
between genes and the environment over time.

Citation source: Molecular Psychiatry 2004 Volume 9, advance on line
publication doi:10.1038/sj.mp.4001529

For further information on this work, please contact Mady Hornig, MD,
Columbia University, Mailman School of Public Health, Greene Infectious
Disease Laboratory, 722 W 168th St, New York, New York 10032, United States
of America, phone: 212-342-9036; FAX: 949-824-1229; e-mail:


ARTICLE: "Neurotoxic effects of postnatal thimerosal are mouse
strain-dependent"

M Hornig, D Chian, W. I. Lipkin

Greene Infectious Disease Laboratory, Mailman School of Public Health,
Columbia University, 722 W 168th St, New York, New York 10032

http://www.ncbi.nlm.nih.gov/entrez/q..._uids=15527868

1: Neurotoxicology. 2005 Jan;26(1):1-8. Related Articles, Links


Thimerosal neurotoxicity is associated with glutathione depletion:
protection with glutathione precursors.

James SJ, Slikker W 3rd, Melnyk S, New E, Pogribna M, Jernigan S.

Department of Pediatrics, University of Arkansas for Medical Sciences and
Arkansas Children's Hospital Research Institute, Little Rock, AR 72202, USA.


Thimerosol is an antiseptic containing 49.5% ethyl mercury that has been
used for years as a preservative in many infant vaccines and in flu
vaccines. Environmental methyl mercury has been shown to be highly
neurotoxic, especially to the developing brain. Because mercury has a high
affinity for thiol (sulfhydryl (-SH)) groups, the thiol-containing
antioxidant, glutathione (GSH), provides the major intracellular defense
against mercury-induced neurotoxicity. Cultured neuroblastoma cells were
found to have lower levels of GSH and increased sensitivity to thimerosol
toxicity compared to glioblastoma cells that have higher basal levels of
intracellular GSH. Thimerosal-induced cytotoxicity was associated with
depletion of intracellular GSH in both cell lines. Pretreatment with 100
microM glutathione ethyl ester or N-acetylcysteine (NAC), but not
methionine, resulted in a significant increase in intracellular GSH in both
cell types. Further, pretreatment of the cells with glutathione ethyl ester
or NAC prevented cytotoxicity with exposure to 15 microM Thimerosal.
Although Thimerosal has been recently removed from most children's vaccines,
it is still present in flu vaccines given to pregnant women, the elderly,
and to children in developing countries. The potential protective effect of
GSH or NAC against mercury toxicity warrants further research as possible
adjunct therapy to individuals still receiving Thimerosal-containing
vaccinations.

PMID: 15527868 [PubMed - indexed for MEDLINE]

http://www.ncbi.nlm.nih.gov/entrez/q..._uids=15184908

Mol Psychiatry. 2004 Sep;9(9):833-45. Related Articles, Links


Neurotoxic effects of postnatal thimerosal are mouse strain dependent.

Hornig M, Chian D, Lipkin WI.

Jerome L and Dawn Greene Infectious Disease Laboratory, Department of
Epidemiology, Mailman School of Public Health, Columbia University, New
York, NY 10032, USA.

The developing brain is uniquely susceptible to the neurotoxic hazard posed
by mercurials. Host differences in maturation, metabolism, nutrition, sex,
and autoimmunity influence outcomes. How population-based variability
affects the safety of the ethylmercury-containing vaccine preservative,
thimerosal, is unknown. Reported increases in the prevalence of autism, a
highly heritable neuropsychiatric condition, are intensifying public focus
on environmental exposures such as thimerosal. Immune profiles and family
history in autism are frequently consistent with autoimmunity. We
hypothesized that autoimmune propensity influences outcomes in mice
following thimerosal challenges that mimic routine childhood immunizations.
Autoimmune disease-sensitive SJL/J mice showed growth delay; reduced
locomotion; exaggerated response to novelty; and densely packed,
hyperchromic hippocampal neurons with altered glutamate receptors and
transporters. Strains resistant to autoimmunity, C57BL/6J and BALB/cJ, were
not susceptible. These findings implicate genetic influences and provide a
model for investigating thimerosal-related neurotoxicity.

PMID: 15184908 [PubMed - indexed for MEDLINE]

http://poisonevercure.150m.com/autism.htm

Autistic children are shown to retain abnormally high concentrations of
mercury from environmental sources such as vaccines.

********* (Until recently, the FDA administration concealed their knowledge
that thimerosal has been known to cross through the blood-brain barrier and
concentrate in the brain).***********

In a recent communication with Congressman Dr. Weldon, CDC conceded that
some of the routinely recommended vaccines contained the full amount of
thimerosal (25 mcg) as late as 2003. Those are not to expire until towards
the end of 2005. There is no existing reason to believe that manufactures
have it in mind to completely remove thimerosal from childhood vaccines in
the near future. Much to my alarm, documents recently obtained from the
World Health Organization (WHO)state that their policy is to lobby strongly
for maintaining thimerosal in vaccines as they see it necessary to use
childhood vaccines in third world countries. The mentality is that if
thimerosal is taken out of American childhood vaccines, the third world
countries will not accept thimerosal-containing childhood vaccines. This
seems to be a clear disturbing indication that, for whatever reason, WHO
desires to inoculate third world country populations with thimerosal
containing vaccines. This is an agency that claims to have an interest in
making sure that children in developing countries have the best
opportunities at life. How is that possible when they are being
deliberately poisoned with high concentrations of a neurotoxins?
There exists many decades worth of peer-reviewed literature (literally
hundreds) on the dangers of thimerosal which include case-reports, animal
studies, tissues culture studies, genetic studies, toxicology studies, and
biochemical studies. According to the above article, CDC, HHS and AAP warns
that 1/166 children have autistic spectrum disorders and even more alarming,
1/6 children have developmental and or behavioral disorders.
The World Health Organization's (WHO) Expert Committee on Biological
Standardization acknowledges that thimerosal is essential during vaccine
production to inactivate certain pathogenic organisms and toxins and prevent
microbial growth during vaccine storage and use. (click here to view
document). Read the Eli Lilly's, manufacturer of thimerosal, safety data
sheet on thimerosal. According to this document, thimerosal will react with
strong oxidizing agents and one listed is peroxides. Another vaccine
component. Also listed are the effects, including signs and symptoms of
exposure such as topical allergic dermatitis, topical hypersensitivity
reactions. Early signs of mercury poisoning are noted as nervous system
effects which include narrowing of the visual field and numbness in the
extremities. "Exposure to mercury in utero and in children can cause mild
to severe mental retardation and mild to severe motor coordination's
impairment". Primary routes of entry are listed as inhalation and skin
contact. For shipping information, there's no question of the label:
POISONS accompanied by the skull and bones picture label.
Mercury over stimulates the brain's immune system. Over stimulation of the
brain results in activation of the microglia widely dispersed in the brain.
When the microglia are activated, they release toxins killing surrounding
brains cells. Prolonged stimulation of the microglia by too many vaccines
kills far too many brain cells.
Though, some may find the reasoning of this imitation form of immunization
to make sense and logic, studying the peer review, lab work and studies
conducting the safety of such the practice will encourage you to think
twice. The dangers of inoculating children and adults with vile
microorganisms is potentially fatal. World Health Organization is privy to
this information. Other material indicate they know that more children
would die and or die quicker without the thimerosal. Sounds insane, but a
fact worth keeping in mind and or researching on your own. So, in order to
inactivate these microorganisms something even more toxic is needed to do
just that. That's where the thimerosal comes in. These facts alone should
raise a few eyebrows. Remember, in the records of mercury toxicology, it
only takes 35 mcg to kill a rabbit. Now, think about how much is in each
vaccine. There's 25mcg in Hib, Pneumococcal (except for Prevnar), DTaP, all
Tetanus brands. Then there's 12.5 in the Hep b. How much thimerosal is
needed should be your other indicator of the dangers of vaccines. The next
indicator is how many doses children receive by school registration.
It's one Russian roulette game after another to keep the big bucks packing
into the pockets of the big dogs.

Mol Psychiatry. 2004 Sep;9(9):833-45.Related Articles, Links


Neurotoxic effects of postnatal thimerosal are mouse strain dependent.

Hornig M, Chian D, Lipkin WI.

Jerome L and Dawn Greene Infectious Disease Laboratory, Department of
Epidemiology, Mailman School of Public Health, Columbia University, New
York, NY 10032, USA.

The developing brain is uniquely susceptible to the neurotoxic hazard posed
by mercurials. Host differences in maturation, metabolism, nutrition, sex,
and autoimmunity influence outcomes. How population-based variability
affects the safety of the ethylmercury-containing vaccine preservative,
thimerosal, is unknown. Reported increases in the prevalence of autism, a
highly heritable neuropsychiatric condition, are intensifying public focus
on environmental exposures such as thimerosal. Immune profiles and family
history in autism are frequently consistent with autoimmunity. We
hypothesized that autoimmune propensity influences outcomes in mice
following thimerosal challenges that mimic routine childhood immunizations.
Autoimmune disease-sensitive SJL/J mice showed growth delay; reduced
locomotion; exaggerated response to novelty; and densely packed,
hyperchromic hippocampal neurons with altered glutamate receptors and
transporters. Strains resistant to autoimmunity, C57BL/6J and BALB/cJ, were
not susceptible. These findings implicate genetic influences and provide a
model for investigating thimerosal-related neurotoxicity.

PMID: 15184908 [PubMed - indexed for MEDLINE]

1: Neurotoxicology. 1989 Winter;10(4):699-706.Related Articles, Links


Effect of organic and inorganic mercuric salts on Na+K+ATPase in different
cerebral fractions in control and intrauterine growth-retarded rats:
alterations induced by serotonin.

Chanez C, Flexor MA, Bourre JM.

Unite 26, INSERM, Hopital Fernand WIDAL, Paris, France.

An intrauterine growth-retarded (IUGR) model based on restriction of blood
supply to the rat fetus at the 17th day of pregnancy was studied. We
investigated in vitro the effects of thimerosal and mercuric chloride on
Na+K+ATPase activity in total brain homogenate, synaptosomes and myelin at
weaning. In addition, we evaluated the reversal effect of serotonin on
mercury-inhibited Na+K+ATPase activity. The toxicity, in terms of inhibition
of Na+K+ATPase activity was greater with mercuric chloride than with
thimerosal. Synaptosomes and principally myelin were more sensitive to the
metal salts than total homogenate. Serotonin stimulated the Na+K+ATPase
activity in total brain homogenate and synaptosomes but inhibited the enzyme
in the myelin fraction. This effect was more marked in the IUGR group than
in the control group. Serotonin (1 mM) added to total homogenate pretreated
with the mercury salts produced variable reversal effects. In the
synaptosomal fraction reverse effect was noted with serotonin. In myelin
fraction, added serotonin increased inhibition caused by thimerosal.

PMID: 2562765 [PubMed - indexed for MEDLINE]

1: Int J Biochem. 1983;15(1):5-7.Related Articles, Links


Rat brain (Na+-K+)ATPase: modulation of its ouabain-sensitive K+-PNPPase
activity by thimerosal.

Lewis RN, Bowler K.

1. The (Na+ + K+) ATPase activity of a rat brain synaptic membrane
preparation was inhibited by 10(-5) M thimerosal. 2. The ouabain inhibitable
K+-PNPPase activity of thimerosal treated membranes was compared with that
of untreated membranes with respect to sensitivity to temperature, ouabain,
K+ and ATP. 3. All those kinetic characteristics were substantially altered
by treatment with thimerosal.

PMID: 6298022 [PubMed - indexed for MEDLINE]

pharmacist friend maintains multiple dose vials of flu vaccine all
contain mercury as a preservative/antibacterial. the single dose vials
do not have mercury as a preservative, but have had mercury added
during the initial processing of the vaccine. the resultant single
dose vials have a minute amount of mercury, esp in comparison to the
multiple dose vials. one can request one's physician order single
dose vial flu vaccine. It is more expensive.
(2-5-04) BOSTON, Mass. - According to new research from Northeastern
University pharmacy professor Richard Deth and colleagues from the
University of Nebraska, Tufts, and Johns Hopkins University, there is
an apparent link between exposure to certain neurodevelopmental toxins
and an increased possibility of developing neurological disorders
including autism and attention-deficit hyperactivity disorder. The
research - the first to offer an explanation for possible causes of
two increasingly common childhood neurological disorders - is
published today in the April 2004 issue of the journal Molecular
Psychiatry.

Though some speculation exists regarding this link, Deth and his
colleagues found that exposure to toxins, such as ethanol and heavy
metals (including lead, aluminum and the ethylmercury-containing
preservative thimerosal) potently interrupt growth factor signaling,
causing adverse effects on methylation reactions (i.e. the transfer of
carbon atoms). Methylation, in turn, plays a significant role in
regulating normal DNA function and gene expression, and is critical to
proper neurological development in infants and children. Scientists and
practitioners have identified an increase in diagnoses of autism and
ADHD in particular, though the reasons why are largely unknown.

In their work, the scientists found that insulin-like growth factor-1
(IGF-1) and the neurotransmitter dopamine both stimulated
folate-dependent methylation pathways in neuronal cells. At the same
time they noted that compounds like thimerosal, ethanol and metals
(like lead and mercury) effectively inhibited these same biochemical
pathways at concentrations that are typically found following
vaccination or other sources of exposure. By better understanding what
happens when infants and children are exposed to these materials, the
work of Deth and his colleagues helps to explain how environmental
contact with metals and administration of certain vaccines may lead to
serious disorders that manifest themselves during childhood, including
autism and ADHD.

"Scientists certainly acknowledge that exposure to neurotoxins like
ethanol and heavy metals can cause developmental disorders, but until
now, the precise mechanisms underlying their toxicity have not been
known," said Deth. "The recent increase in the incidence of autism
led us to speculate that environmental exposures, including vaccine
additives might contribute to the triggering of this disorder."

Thimerosal, which was largely phased out in the U.S. and in Europe
starting in 2000,was often used for its preservative abilities in
multi-dose units of vaccines for diseases like hepatitis, whooping
cough, tetanus and diptheria. Today, most vaccines carry only trace
amounts of it, according to the CDC. But in larger, multi-dose vials of
these vaccines, often shipped to and used in third world countries,
thimerosal is still very common. Multi-dose flu vaccines still contain
thimerosal.

Additionally, the scientists recently obtained more insight into the
mechanism by which thimerosal interferes with folate-dependent
methylation. It acts by inhibiting the biosynthesis of the active form
of vitamin B12 (methylcobalamin), which is of particular interest
because doctors treating autistic kids are having good success with the
administration of methycobalamin.

Northeastern University, a private research institution located in
Boston, Massachusetts, is a world leader in practice-oriented
education. Building on its flagship cooperative education program,
Northeastern links classroom learning with workplace experience and
integrates professional preparation with study in the liberal arts and
sciences. U.S. News & World Report, in its annual guide America's
Best Colleges, 2003, ranked Northeastern University number one in the
country among programs that "require or encourage students to apply
what they're learning in the classroom out in the real world." In
addition, Northeastern's career services was top ranked by Kaplan
Newsweek's "Unofficial Insiders Guide to the 320 Most Interesting
Colleges and Universities," 2003 edition. For more information, please
visit http://www.northeastern.edu.

Paper in full at this link:

http://www.nupr.neu.edu/2-04/deth_article.pdf
Mol Psychiatry. 2004 Apr;9(4):358-70. Related Articles, Links


Activation of methionine synthase by insulin-like growth factor-1 and
dopamine: a target for neurodevelopmental toxins and thimerosal.

Waly M, Olteanu H, Banerjee R, Choi SW, Mason JB, Parker BS, Sukumar S,
Shim S, Sharma A, Benzecry JM, Power-Charnitsky VA, Deth RC.

Department of Pharmaceutical Sciences, Northeastern University, Boston,
MA 02115, USA.

Methylation events play a critical role in the ability of growth
factors to promote normal development. Neurodevelopmental toxins, such
as ethanol and heavy metals, interrupt growth factor signaling, raising
the possibility that they might exert adverse effects on methylation.
We found that insulin-like growth factor-1 (IGF-1)- and
dopamine-stimulated methionine synthase (MS) activity and
folate-dependent methylation of phospholipids in SH-SY5Y human
neuroblastoma cells, via a PI3-kinase- and MAP-kinase-dependent
mechanism. The stimulation of this pathway increased DNA methylation,
while its inhibition increased methylation-sensitive gene expression.
Ethanol potently interfered with IGF-1 activation of MS and blocked its
effect on DNA methylation, whereas it did not inhibit the effects of
dopamine. Metal ions potently affected IGF-1 and dopamine-stimulated MS
activity, as well as folate-dependent phospholipid methylation: Cu(2+)
promoted enzyme activity and methylation, while Cu(+), Pb(2+), Hg(2+)
and Al(3+) were inhibitory. The ethylmercury-containing preservative
thimerosal inhibited both IGF-1- and dopamine-stimulated methylation
with an IC(50) of 1 nM and eliminated MS activity. Our findings outline
a novel growth factor signaling pathway that regulates MS activity and
thereby modulates methylation reactions, including DNA methylation. The
potent inhibition of this pathway by ethanol, lead, mercury, aluminum
and thimerosal suggests that it may be an important target of
neurodevelopmental toxins.

PMID: 14745455 [PubMed - in process]

Medical News Today
Thimerosal, found in childhood vaccines, can increase the risk of
autism-like damage in mice
09 Jun 2004

A new study indicates that postnatal exposure to thimerosal, a mercury
preservative commonly used in a number of childhood vaccines, can lead to
the development of autism-like damage in autoimmune disease susceptible
mice. This animal model, the first to show that the administration of
low-dose ethylmercury can lead to behavioral and neurological changes in
the developing brain, reinforces previous studies showing that a genetic
predisposition affects risk in combination with certain environmental
triggers. The study was conducted by researchers at the Jerome L. and Dawn
Greene

Infectious Disease Laboratory at the Mailman School of Public Health,
Columbia University. Over the past 20 years, there has been a striking
increase--at least ten-fold since 1985--in the number of children
diagnosed with autism spectrum disorders. Genetic factors alone cannot
account for this rise in prevalence. Researchers at the Mailman School,
led by Dr. Mady Hornig, created an animal model to explore the
relationship between thimerosal (ethylmercury) and autism, hypothesizing
that the combination of genetic susceptibility and environmental exposure
to mercury in childhood vaccines may cause neurotoxicity.

Cumulative mercury burden through other sources, including in utero
exposures to mercury in fish or vaccines, may also lead to damage in
susceptible hosts. Timing and quantity of thimerosal dosing for the mouse
model were developed using the U.S. immunization schedule for children,
with doses calculated for mice based on 10th percentile weight of U.S.
boys at age two, four, six, and twelve months.

The researchers found the subset of autoimmune disease susceptible mice
with thimerosal exposure to express many important aspects of the
behavioral and neuropathologic features of autism spectrum disorders,
including:

Abnormal response to novel environments;

Behavioral impoverishment (limited range of behaviors and decreased
exploration of environment); Significant abnormalities in brain
architecture, affecting areas subserving emotion and cognition; Increased
brain size.

These findings have relevance for identification of autism cases relating
to environmental factors; design of treatment strategies; and development
of rational immunization programs. The use of thimerosal in vaccines has
been reduced over the past few years, although it is still present in some
influenza vaccines. Identifying the connection between genetic
susceptibility and an environmental trigger for autism--in this case
thimerosal exposure--is important because it may promote discovery of
effective interventions for and limit exposure in a specific population,
stated the lead author Dr. Mady Hornig. Because the developing brain can
be exposed to toxins that are long gone by the time symptoms appear, clues
gathered in these animal models can then be evaluated through prospective
human birth cohorts--providing a powerful to tool to dissect the
interaction between genes and the environment over time.

Citation source: Molecular Psychiatry 2004 Volume 9, advance on line
publication doi:10.1038/sj.mp.4001529

For further information on this work, please contact Mady Hornig, MD,
Columbia University, Mailman School of Public Health, Greene Infectious
Disease Laboratory, 722 W 168th St, New York, New York 10032, United
States of America, phone: 212-342-9036; FAX: 949-824-1229; e-mail:


http://www.altcorp.com/DentalInformation/asdexperts.htm

http://www.nupr.neu.edu/2-04/deth_article.pdf

Mol Psychiatry. 2004 Apr;9(4):358-70. Related Articles, Links


Activation of methionine synthase by insulin-like growth factor-1 and
dopamine: a target for neurodevelopmental toxins and thimerosal.

Waly M, Olteanu H, Banerjee R, Choi SW, Mason JB, Parker BS, Sukumar S,
Shim S, Sharma A, Benzecry JM, Power-Charnitsky VA, Deth RC.

Department of Pharmaceutical Sciences, Northeastern University, Boston,
MA 02115, USA.

Methylation events play a critical role in the ability of growth
factors to promote normal development. Neurodevelopmental toxins, such
as ethanol and heavy metals, interrupt growth factor signaling, raising
the possibility that they might exert adverse effects on methylation.
We found that insulin-like growth factor-1 (IGF-1)- and
dopamine-stimulated methionine synthase (MS) activity and
folate-dependent methylation of phospholipids in SH-SY5Y human
neuroblastoma cells, via a PI3-kinase- and MAP-kinase-dependent
mechanism. The stimulation of this pathway increased DNA methylation,
while its inhibition increased methylation-sensitive gene expression.
Ethanol potently interfered with IGF-1 activation of MS and blocked its
effect on DNA methylation, whereas it did not inhibit the effects of
dopamine. Metal ions potently affected IGF-1 and dopamine-stimulated MS
activity, as well as folate-dependent phospholipid methylation: Cu(2+)
promoted enzyme activity and methylation, while Cu(+), Pb(2+), Hg(2+)
and Al(3+) were inhibitory. The ethylmercury-containing preservative
thimerosal inhibited both IGF-1- and dopamine-stimulated methylation
with an IC(50) of 1 nM and eliminated MS activity. Our findings outline
a novel growth factor signaling pathway that regulates MS activity and
thereby modulates methylation reactions, including DNA methylation. The
potent inhibition of this pathway by ethanol, lead, mercury, aluminum
and thimerosal suggests that it may be an important target of
neurodevelopmental toxins.

Thimerosal neurotoxicity and protection with
N-Acetylcysteine supplementation", Health Sentinel, January 3, 2005,

In the 1930s, Eli Lily developed Thimerosal as a preservative and it
was widely used in vaccines. Until the removal of Thimerosal, which
contains 49.9% ethyl mercury by weight, from most pediatric vaccines in
2001, the source of the largest human exposure to mercury in the US was
in children under 18 months of age undergoing routine childhood
immunization schedules. Before 2001, a child may have received a
cumulative dose of over 200 µg/kg (micrograms per kilogram) in the
first 18 months of life.

Although Thimerosal has been removed from most childhood vaccines, it
is still present in the flu vaccine, which is given to pregnant women,
the elderly, and children. Also, many vaccines given to children in
developing countries still contain Thimerosal.

In the 2005 issue of NeuroToxicology, the authors of a study examine
the toxicity of Thimerosal within the body including neurons. They
examine the neurotoxic mechanisms, how the body detoxifies mercury, and
the use of N-Acetylcysteine, or NAC for short, in facilitating the
detoxification pathway within the body.

Glutathione, a tripeptide composed of cysteine, glutamate, and glycine,
is manufactured in the liver and also in the brain. Normally, the
concentrations of glutathione in the cells are quite high providing for
detoxification of a variety of heavy metals including mercury. However,
when this essential antioxidant is depleted the excess mercury can bind
to internal cellular proteins leading to toxic damage. Studies have
shown that, "low micromolar concentrations of Thimerosal induced DNA
strand breaks, caspase-3 activation, membrane damage and cell death."

Although the brain can produce glutathione, it can only manufacture
this from its immediate precursor cysteine. The liver, on the other
hand, is able through a long series of biochemical steps to create
glutathione from methionine. Methionine is an essential amino acid that
supplies the body with sulfur and methyl groups. The liver uses a
number of enzyme systems along with various B vitamins to produce
glutathione. The liver then exports the glutathione to the blood that
then is broken down to cystine. Cystine crosses the blood-brain barrier
to be used by the brain to make glutathione. Thus, the brain is reliant
on the liver to manufacture chemicals to keep it free from toxins.

The brain contains neurons and other cells called astrocytes.
Astrocytes use the cystine that crosses the blood-brain barrier to make
glutathione. The astrocytes then export the glutathione to the space
between the cells where it is broken down to cysteine. The neurons take
up the cysteine and manufacture glutathione. This complex series of
biochemical events is what is necessary to keep the brain free from
heavy metal damage.

The authors first examined the level of Thimerosal that would cause
toxic damage to cells. They found that the higher the concentration of
Thimerosal the greater the number of cells that were killed although
the nerve cell response occurred with only a 3 hour exposure, whereas
the other cell line required a 48 hour exposure demonstrating that
nerve cells are more sensitive to Thimerosal toxicity. "In both cell
lines, a progressive increase in cytotoxicity (decrease in viability)
was observed when Thimerosal dose was progressively doubled from 2.5
µmol/L [micromoles per liter] to 5, 10, and 20 µmol/L. Viability was
reduced more than 50% in both cell lines with exposure to 10 µmol/L
Thimerosal and less than 10% of cells survived a dose of 20 µmol/L."

The authors then pretreated cells with NAC before adding a dose of 15
µmol/L Thimerosal. They found that, "Thimerosal alone induced more
than a 6-fold decrease in viability", and that NAC, "provided
significant protection against cell death". The authors note,
"Thimerosal induces oxidative stress and apoptosis by activating
mitochondrial cell death pathways. A subsequent study using cultured
human neuron and fibroblast cell lines similarly showed that low
micromolar concentrations of Thimerosal induced DNA strand breaks,
caspase-3 activation, membrane damage and cell death."

The authors conclude that, "numerous clinical studies have
demonstrated the efficacy of NAC in increasing intracellular
glutathione levels and reducing oxidative stress in humans. Since
cytotoxicity with both ethyl- and methyl- mercury have been shown to be
mediated by glutathione depletion, dietary supplements that increase
intracellular glutathione could be envisioned as an effective
intervention to reduce previous or anticipated exposure to mercury.
This approach would be especially valuable in the elderly and in
pregnant women receiving Rho D immunoglobulin shots, and individuals
who regularly consume mercury-containing fish."

SOURCE: NeuroToxicology, Vol. 26, 2005, pp. 1-8

http://www.childproofing.org/vaccine&autism.html

New Research Suggests Link Between Vaccine Ingredients and Autism, ADHD
[Source: Northeastern University.]
http://www.newswise.com/articles/view/503041/NewsWise

Newswise - According to new research from Northeastern University pharmacy
professor Richard Deth and colleagues from the University of Nebraska,
Tufts, and Johns Hopkins University, there is an apparent link between
exposure to certain neurodevelopmental toxins and an increased possibility
of developing neurological disorders including autism and attention-deficit
hyperactivity disorder. The research - the first to offer an explanation for
possible causes of two increasingly common childhood neurological
disorders - is published today in the April 2004 issue of the journal
Molecular Psychiatry.

Though some speculation exists regarding this link, Deth and his colleagues
found that exposure to toxins, such as ethanol and heavy metals (including
lead, aluminum and the ethylmercury-containing preservative thimerosal)
potently interrupt growth factor signaling, causing adverse effects on
methylation reactions (i.e. the transfer of carbon atoms). Methylation, in
turn, plays a significant role in regulating normal DNA function and gene
expression, and is critical to proper neurological development in infants
and children. Scientists and practitioners have identified an increase in
diagnoses of autism and ADHD in particular, though the reasons why are
largely unknown.

In their work, the scientists found that insulin-like growth factor-1(IGF-1)
and the neurotransmitter dopamine both stimulated folate-dependent
methylation pathways in neuronal cells. At the same time they noted that
compounds like thimerosal, ethanol and metals (like lead and mercury)
effectively inhibited these same biochemical pathways at concentrations that
are typically found following vaccination or other sources of exposure.

By better understanding what happens when infants and children are exposed
to these materials, the work of Deth and his colleagues helps to explain how
environmental contact with metals and administration of certain vaccines may
lead to serious disorders that manifest themselves during childhood,
including autism and ADHD.

"Scientists certainly acknowledge that exposure to neurotoxins like ethanol
and heavy metals can cause developmental disorders, but until now, the
precise mechanisms underlying their toxicity have not been known," said
Deth. "The recent increase in the incidence of autism led us to speculate
that environmental exposures, including vaccine additives might contribute
to the triggering of this disorder."

Thimerosal, which was largely phased out in the U.S. and in Europe starting
in 2000, was often used for its preservative abilities in multi-dose units
of vaccines for diseases like hepatitis, whooping cough, tetanus and
diptheria.

Today, most vaccines carry only trace amounts of it, according to the CDC.
But in larger, multi-dose vials of these vaccines, often shipped to and used
in third world countries, thimerosal is still very common. Multi-dose flu
vaccines still contain thimerosal.

Additionally, the scientists recently obtained more insight into the
mechanism by which thimerosal interferes with folate-dependent methylation.

It acts by inhibiting the biosynthesis of the active form of vitamin B12
(methylcobalamin), which is of particular interest because doctors treating
autistic kids are having good success with the administration of
methycobalamin.

http://www.altcorp.com/DentalInformation/thimerosal.htm

Medical News Today
Thimerosal, found in childhood vaccines, can increase the risk of
autism-like damage in mice
09 Jun 2004

A new study indicates that postnatal exposure to thimerosal, a mercury
preservative commonly used in a number of childhood vaccines, can lead to
the development of autism-like damage in autoimmune disease susceptible
mice. This animal model, the first to show that the administration of
low-dose ethylmercury can lead to behavioral and neurological changes in
the developing brain, reinforces previous studies showing that a genetic
predisposition affects risk in combination with certain environmental
triggers. The study was conducted by researchers at the Jerome L. and Dawn
Greene

Infectious Disease Laboratory at the Mailman School of Public Health,
Columbia University. Over the past 20 years, there has been a striking
increase--at least ten-fold since 1985--in the number of children
diagnosed with autism spectrum disorders. Genetic factors alone cannot
account for this rise in prevalence. Researchers at the Mailman School,
led by Dr. Mady Hornig, created an animal model to explore the
relationship between thimerosal (ethylmercury) and autism, hypothesizing
that the combination of genetic susceptibility and environmental exposure
to mercury in childhood vaccines may cause neurotoxicity.

Cumulative mercury burden through other sources, including in utero
exposures to mercury in fish or vaccines, may also lead to damage in
susceptible hosts. Timing and quantity of thimerosal dosing for the mouse
model were developed using the U.S. immunization schedule for children,
with doses calculated for mice based on 10th percentile weight of U.S.
boys at age two, four, six, and twelve months.

The researchers found the subset of autoimmune disease susceptible mice
with thimerosal exposure to express many important aspects of the
behavioral and neuropathologic features of autism spectrum disorders,
including:

Abnormal response to novel environments;

Behavioral impoverishment (limited range of behaviors and decreased
exploration of environment); Significant abnormalities in brain
architecture, affecting areas subserving emotion and cognition; Increased
brain size.

These findings have relevance for identification of autism cases relating
to environmental factors; design of treatment strategies; and development
of rational immunization programs. The use of thimerosal in vaccines has
been reduced over the past few years, although it is still present in some
influenza vaccines. Identifying the connection between genetic
susceptibility and an environmental trigger for autism--in this case
thimerosal exposure--is important because it may promote discovery of
effective interventions for and limit exposure in a specific population,
stated the lead author Dr. Mady Hornig. Because the developing brain can
be exposed to toxins that are long gone by the time symptoms appear, clues
gathered in these animal models can then be evaluated through prospective
human birth cohorts--providing a powerful to tool to dissect the
interaction between genes and the environment over time.

Citation source: Molecular Psychiatry 2004 Volume 9, advance on line
publication doi:10.1038/sj.mp.4001529

For further information on this work, please contact Mady Hornig, MD,
Columbia University, Mailman School of Public Health, Greene Infectious
Disease Laboratory, 722 W 168th St, New York, New York 10032, United
States of America, phone: 212-342-9036; FAX: 949-824-1229; e-mail:

I am aware of the points that you make, Dorothy; however, I do not buy
that these are the only explanations. I don't discredit them though
either. I definitely don't accept that the reason the diagnosis age-
range begins at 18 months is due to when parents "notice" a difference
compared to other children at all. Having an 18-month-old myself
currently, 18-months is about the age, give or take, that the children
really blossom into the running and communicating animals they are -
God bless 'em. lol. When you say "look at them before their first
birthday," it does nothing to change the fact that their little
rapidly developing immune systems are assaulted with vaccine after
vaccine, which happen to contain toxic substances, beginning with the
first vaccine being administered within 48 hours of birth - so yes,
these children MAY just be different prior to that first birthday and
for a reason that nobody wishes to look into further. I pray that
someone would wipe these thoughts out of my head with at least an
attempt, but they don't. I've pointed it out before and will again. I
am not anti-vaccine and my children have received them. I do really
wish they did nothing but good, but they don't. They do harm some
people, and THAT warrants more than a looksee. I just don't believe
they should even be offered to me without providing me with all of the
necessary information I need to make an educated decision as to
whether or not the risks really do outweight he benefits in OUR
situation. I believe that if the answer is something simple like "wait
until their immune systems are more developed before beginning," then
we should. I'm not even here to say that I wholeheartedly support John
in his stance on the issue, but I do support the fact that each post
can bring to light that vaccines are not something you should just
"give" your child because your doctor told you that you should. People
need to be made aware that each and every vaccine administered to
their children warrants at least a thought, and even a prayer, that
their child won't be the one harmed as a result.


On Apr 15, 11:03�am, toto wrote:
On 14 Apr 2007 22:10:03 -0700, "Chris" wrote:

I'm curious as to how you know this to be fact Jeff. One of the stats
I saw on TV the other day I found interesting - 1 in 97 boys will be
diagnosed with some form of autism and that the ages of diagnosis or
onset is from something like 18 months to 6 years. Isn't that
precisely when these children, or at least just after that first tough
year of the multitude of vaccines, receive all the majority of their
vaccines for school entry?

First of all, the incidence of autism has increased as the incidence
of mental retardation and other diagnoses has decreased. *Part of the
increase, then is due to better understanding of autism and better
diagnoses.

Second, the incidence increased because the diagnostic standards have
changed to include PDD-NOS and Asperger's which were not diagnosable
until around 1990.

Third, most autism is *not* the regressive kind of autism. *For the
children I know, most did not regress, they simply did not progress in
the way that *normal* children did. *The disorder becomes more
noticeable at around 18 months because that is when most parents see
the differences from the NT children of their friends. *Many first
time parents don't notice these differences at all until children hit
preschool and they see how many quirks their children have in
comparison to NT three year olds. *My grandson was *in his own world*
probably from the very beginning of his life.

Fourth, the fact that there is a correlation between time of noticing
onset and time of vaccination does not mean that the vaccines were the
cause of the condition. *

--
Dorothy

There is no sound, no cry in all the world
that can be heard unless someone listens ..

The Outer Limits

On Apr 15, 3:23?pm, "Jeff" wrote:
"Chris" wrote in message

ups.com...



Actually, the number is 0.

I'm curious as to how you know this to be fact Jeff. One of the stats
I saw on TV the other day I found interesting - 1 in 97 boys will be
diagnosed with some form of autism and that the ages of diagnosis or
onset is from something like 18 months to 6 years. Isn't that
precisely when these children, or at least just after that first tough
year of the multitude of vaccines, receive all the majority of their
vaccines for school entry?

yes, but if you compare the videos of kids before their first birthdays who
later develop autism to those who don't, you can see differences in the
behavior of the two sets kids, especially social behaviors (this is done in
a blind manner, meaning that the people who are watching the kids don't know
which ones will become autistics later).

All of the evidence indicates that vaccination does not cause autism.

Jeff

Keep in mind, that I nowhere refer to the mercury and autism debate,
because I don't just think of mercury. The fact that the normal immune
system is bypassed in the process of a vaccine, the fact that autism
is "a disorder probably caused by organically based central nervous
system dysfunction", the fact that the neurologic systems, immunologic
systems, etc. are rapidly forming during a period of time when an
infant is supposed to receive nothing other than breastmilk, the fact
that a body's immune system can be confused at just what it should be
attacking and thereby attack itself bringing on the diagnosis of an
autoimmune disease, warrants research in a direction most involved
prefer to ignore.

What would you do if faced with the following decision to make: You
have to choose either taking your chances outside on your own or
taking a small risk inside a protected environment -- you and your
infant for 30 days --- outside you don't know what could happen, if or
when you may contract a condition from which you may or may not
recover fully from, live by your own resources, etc. and inside
everything you need is provided for you without worry to you. However,
inside, with the selected 30,000 people, 1 single individual has a
contagious disease that you may or may not fully recover from. You are
told not to worry about that 1 individual because your chances of
contracting the condition is only 1 in 30,000. Which do you choose?

"Chris" wrote in message
oups.com...
On Apr 15, 3:23?pm, "Jeff" wrote:
"Chris" wrote in message

ups.com...



Actually, the number is 0.

I'm curious as to how you know this to be fact Jeff. One of the stats
I saw on TV the other day I found interesting - 1 in 97 boys will be
diagnosed with some form of autism and that the ages of diagnosis or
onset is from something like 18 months to 6 years. Isn't that
precisely when these children, or at least just after that first tough
year of the multitude of vaccines, receive all the majority of their
vaccines for school entry?

yes, but if you compare the videos of kids before their first birthdays
who
later develop autism to those who don't, you can see differences in the
behavior of the two sets kids, especially social behaviors (this is done
in
a blind manner, meaning that the people who are watching the kids don't
know
which ones will become autistics later).

All of the evidence indicates that vaccination does not cause autism.

Jeff

Keep in mind, that I nowhere refer to the mercury and autism debate,
because I don't just think of mercury. The fact that the normal immune
system is bypassed in the process of a vaccine,

Actually, the normal immune system is not bypassed, rather it is activited,
although more specifically than with a natural infection.

the fact that autism
is "a disorder probably caused by organically based central nervous
system dysfunction", the fact that the neurologic systems, immunologic
systems, etc. are rapidly forming during a period of time when an
infant is supposed to receive nothing other than breastmilk,

and the immune system receives thousands of other challenges each day, like
from the bacteria in the gut

the fact
that a body's immune system can be confused at just what it should be
attacking and thereby attack itself bringing on the diagnosis of an
autoimmune disease,

autism is not an autoimmune disease, however. To the best of my knowledge,
there is no evidence to suggest that it is.

warrants research in a direction most involved
prefer to ignore.

What direction is that?

Considering that babies are exposed to many different types of pollution,
like air pollution, pesticides, household dirt, different bacteria, viruses
and other micororganisms, vaccines are really not much of a threat compared
to all the other threats.

What would you do if faced with the following decision to make: You
have to choose either taking your chances outside on your own or
taking a small risk inside a protected environment -- you and your
infant for 30 days --- outside you don't know what could happen, if or
when you may contract a condition from which you may or may not
recover fully from, live by your own resources, etc. and inside
everything you need is provided for you without worry to you. However,
inside, with the selected 30,000 people, 1 single individual has a
contagious disease that you may or may not fully recover from. You are
told not to worry about that 1 individual because your chances of
contracting the condition is only 1 in 30,000. Which do you choose?

Going outside. Maybe there is more risk outside, but there is more
opportunity, as well.

Jeff

On Apr 13, 7:54 am, "Jeff" wrote:
"bigvince" wrote in message

oups.com...
...

So all those scrips for Alzheimer's meds are based on what.? A
hunch? and all those studies showing improvement as a result of
treating a disorder that cannot be definitively diagnosed until the
patient dies are based on bogus science.

Please provide a better alternative The current treatments are so ineffective that that bar is not very high. Why kind of diagnoses do you think
"alternative medicine" practioners make? Very poor ones not based on any
type of understanding of the biological basis of disease. Is there *any*
treatment that provide for any meaningful help for Alzheimer's?

The answer is yes. The present treatments give victims of Alzheimer's
disease about 6 more months of cognitive ability. And as researchers better
understand the pathology of Alzheimer's, they are better able to develop new
treatments.
In England Nice guidelines eliminated these drugs as treatment options because of lack of benefit at least to the patient . A Johns Hopkins reseacher asked the same question.

The diagnosis of Alzheimer's disease is based on several factors,
like
history and physical of the patient, and tests that eliminate other possible
causes of cognitive decline. Neurocognitive studies can delineate the
cognitive decline of the patient, helping to make an accurate diagnosis.
Imaging studies like MRIs and PET scans can also be used. The diagnosis is
provisional, unless a brain biopsy is done.

Now, you are saying that this is bogus science. Well, the science shows that
people who are given particular drugs are able to delay their cognitive
decline by about six months. Not great, but this time can be a great gift to
the victims of Alzheimer's. The victim's include the family of the patient.
How is that bogus? The diagnosis is not 100% accurate. But, it turns out
that the diagnoses are very accurate and the data help other patients.
That's good science.
If it is such good science and the drugs are beneficial how come the MDs in Britton concluded the opposite.
BTW, there is a definitive test that can be done while the patient is alive:
brain biopsy.

Here is some information where you can educate yourself about Alzheimer's:http://www.alz.org/alzheimers_diseas...rs_disease.asp

And this is an extremely touching story about a person with Alzheimer's:http://www.npr.org/templates/story/s...toryId=9538242

In my opinion, it is an excellent piece of journalism.

Jeff

Actually, the normal immune system is not bypassed, rather it is activited,
although more specifically than with a natural infection.

You believe that? You believe that the injection for diseases into the
tissues of a body doesn't completely bypass the "normal" immune
process in relation to normal contraction of the diseases? Emphasis on
the word "normal" here. Are you aware of the difference as to what
reactions/processes are evoked between the two introduction methods
and in what order? I am completely aware of this "activation" process
of which you speak.


the fact that autism
is "a disorder probably caused by organically based central nervous
system dysfunction", the fact that the neurologic systems, immunologic
systems, etc. are rapidly forming during a period of time when an
infant is supposed to receive nothing other than breastmilk,

and the immune system receives thousands of other challenges each day, like
from the bacteria in the gut

Yes, but more importantly, naturally, and that bacteria in the gut is
something that the infant's gut is already working hard at adjusting
to, which doesn't even occur until somewhere around an estimated 6
months of age, so why stress a developing organism even more?



the fact
that a body's immune system can be confused at just what it should be
attacking and thereby attack itself bringing on the diagnosis of an
autoimmune disease,

autism is not an autoimmune disease, however. To the best of my knowledge,
there is no evidence to suggest that it is.

I didn't say that autism was an autoimmune disease. You seemed to have
left out the definition of what autism is that I posted in your C&P. I
believe that a developing neurological system can be injured as well.


warrants research in a direction most involved
prefer to ignore.

What direction is that?


You don't really care, so why ask? I've pointed out many times that
the only true way to ensure that vaccines are not in fact harmful for
some people and that it may require further research into who should
and shouldn't receive one or more vaccines is warranted. Would it
really be so terrible to perform a few expensive tests prior to their
administration? Funding for research in this direction isn't readily
available for obvious reasons.



Considering that babies are exposed to many different types of pollution,
like air pollution, pesticides, household dirt, different bacteria, viruses
and other micororganisms, vaccines are really not much of a threat compared
to all the other threats.

You are very strange to believe that my 18-month-old has as much of a
threat from dying or being crippled due to his cold, pinkeye, or
household dirt as he does from a vaccine. I for one do not agree with
any levels of toxins being "within an acceptable range" in my everyday
items or activities of daily living and it happens to be something
else to consider in reference to the rise of ailments affecting people
today. Just because pumping gas can harm me doesn't mean I should take
up smoking. @@


I'm curious Jeff, do you have children?