HepB Vaccine Causes MS Then Death - Vaccine Court Ruling

http://whale.to/vaccine/hepb_vaccine.html

[2009 Jan] HepB Vaccine Causes MS Then Death - Vaccine Court Ruling
Entitlement; Hep B vaccine; two months later, Devic's Disease (a variant of
MS) then death. Petitioner has prevailed on the issue of entitlement. The
medical records during decedent's final hospitalization reflect that she
died from demyelinating disease. Not only did decedent have a vaccine
injury, but also her death was vaccine-related.

JOHN wrote:
http://whale.to/vaccine/hepb_vaccine.html

[2009 Jan] HepB Vaccine Causes MS Then Death - Vaccine Court Ruling
Entitlement; Hep B vaccine; two months later, Devic's Disease (a variant of
MS) then death. Petitioner has prevailed on the issue of entitlement. The
medical records during decedent's final hospitalization reflect that she
died from demyelinating disease. Not only did decedent have a vaccine
injury, but also her death was vaccine-related.



It's worth noting that the victim was an adult woman. She was born in
1961, vaccinated in 1998, got multiple sclerosis and died 10 painful
years later. (Why did she do that? My guess is that it was a requirement
- maybe, she wanted to become a teacher.)

Do not expect Paul Offit say anything about this death.

Mike wrote:

JOHN wrote:
http://whale.to/vaccine/hepb_vaccine.html

[2009 Jan] HepB Vaccine Causes MS Then Death - Vaccine Court Ruling
Entitlement; Hep B vaccine; two months later, Devic's Disease (a variant
of
MS) then death. Petitioner has prevailed on the issue of entitlement.
The medical records during decedent's final hospitalization reflect that
she died from demyelinating disease. Not only did decedent have a vaccine
injury, but also her death was vaccine-related.

It's worth noting that the victim was an adult woman. She was born in
1961, vaccinated in 1998, got multiple sclerosis and died 10 painful
years later. (Why did she do that? My guess is that it was a requirement
- maybe, she wanted to become a teacher.)

Do not expect Paul Offit say anything about this death.

What's to say? Obviously, there was never such a thing as Multiple
Sclerosis prior to the hepatitis B vaccine.

--
| The brighter the stupid burns, the more |
| chance that someone will see the light. |
+- D. C. Sessions -+

"D. C. Sessions" wrote in message
...
Mike wrote:

JOHN wrote:
http://whale.to/vaccine/hepb_vaccine.html

[2009 Jan] HepB Vaccine Causes MS Then Death - Vaccine Court Ruling
Entitlement; Hep B vaccine; two months later, Devic's Disease (a variant
of
MS) then death. Petitioner has prevailed on the issue of entitlement.
The medical records during decedent's final hospitalization reflect that
she died from demyelinating disease. Not only did decedent have a
vaccine
injury, but also her death was vaccine-related.

It's worth noting that the victim was an adult woman. She was born in
1961, vaccinated in 1998, got multiple sclerosis and died 10 painful
years later. (Why did she do that? My guess is that it was a requirement
- maybe, she wanted to become a teacher.)

Do not expect Paul Offit say anything about this death.

What's to say? Obviously, there was never such a thing as Multiple
Sclerosis prior to the hepatitis B vaccine.

Blatant lie.

http://www.protector.pair.com/ms/

Multiple Sclerosis

MS Mystery in Harrison article
Information about Super Blue-Green Algae
History of Multiple Sclerosis
Top Ten Events in MS History

Symptoms of Lindane poisoning compared to symptoms of MS

Lindane poisoning symptom
MS symptom

Seizures Acute flare-ups
Weakness Fatigue
Paresthesia of face and extremities Numbness of extremities
Giddiness Giddiness
Impaired memory Short term memory loss
Loss of sleep Sleeping disorders
Headaches Headaches
Abnormal EEG patterns Abnormal EEG patterns
2/3 women affected 2/3 women affected
Vertigo Dizziness (Erratic walking)
Trauma induced Trauma induced
Loss of libido Loss of sexual urge
Malaise Feeling of discomfort or uneasiness
Tremors Tremors
Apprehension Uneasy, Anxious
Decreased nerve velocity Decreased nerve velocity
Musculoskeletal effects Musculoskeletal dysfunction

As you can easily see above, there are 17 common symptoms listed that almost
exactly match each other. These symptoms lists were taken from the
Toxicological profile for HCH and a medical guide to Multiple Sclerosis.
Some of these symptoms matched word for word. These are the most common
symptoms; there are over 25 less common ones that closely match also.



The History of Multiple Sclerosis
The history of multiple sclerosis (MS) is a detective story spanning more
than a century. Many clues have been pieced together, but only now are
answers emerging. To appreciate why the trail to a solution has been so long
and hard, it is necessary to understand what we scientists now believe to be
true about MS.

Multiple sclerosis is one of the most common diseases of the nervous system,
afflicting people of virtually all ages around the world, although it has a
special preference for young people, especially women, and for those who
grew up in northern latitudes.

We believe MS involves a genetic susceptibility, but it is not directly
inherited. It usually causes sudden neurologic symptoms including vision
loss, paralysis, numbness, and walking difficulties. The symptoms can be
diverse and confusing, often coming and going without any pattern, making it
difficult to diagnose, even today.

The symptoms appear because nerves in the brain and spinal cord lose their
ability to transmit signals. Myelin, a complex substance that surrounds and
insulates nerve fibers, is essential for nerves to conduct electricity and
carry out their function. Myelin is destroyed in MS.

In MS, cells and proteins of the body's immune system, which normally defend
the body against infections, leave the blood vessels serving the central
nervous system, pour into the brain and spinal cord, and destroy myelin. The
specific triggering mechanism which causes an immune system to attack its
own myelin remains unknown, although a viral infection on top of an
inherited genetic susceptibility is a leading suspect.

The discovery of MS

Until the early years of the 19th century, physicians relied on
superstition, hearsay, and the wisdom of the ancients to care for the sick.
Medical ideas were not scientifically tested. Even so, physicians were
sometimes good observers and we can identify people who undoubtedly had MS
from descriptions written as long ago as the Middle Ages. MS has always been
with us.

Once the scientific method took hold in medicine, MS was among the first
diseases to be described scientifically. The 19th-century doctors did not
understand what they saw and recorded, but drawings from autopsies done as
early as 1838 clearly show what we today recognize as MS.

Then, in 1868, Jean-Martin Charcot, a professor of neurology at the
University of Paris, who has been called "the father of neurology",
carefully examined a young woman with a tremor of a sort he had never seen
before. He noted her other neurological problems including slurred speech
and abnormal eye movements, and compared them to those of other patients he
had seen. When she died, he examined her brain and found the characteristic
scars or "plaques" of MS.

Dr. Charcot wrote a complete description of the disease and the changes in
the brain which accompany it. However, he was baffled by its cause and
frustrated by its resistance to all of his treatments. They included
electrical stimulation and strychnine-because this poison is a nerve
stimulant. He also tried injections of gold and silver, as they were
somewhat helpful in the other major nerve disorder common at that
time-syphilis.

A prisoner of biotechnology

In the last decades of the 19th century, the leading physicians of the world
came to understand that MS was a specific disease. MS was recognized in
England by Dr. Moxon in 1873, and in the United States by Dr. Edward Seguin
in 1878. By the end of the century, much of what can be learned about MS
from careful observation was known: that the disease is more common in women
than men, that it is not directly inherited, and that it can produce many
different neurological symptoms.

But observation can go only so far. Knowledge of MS could not advance
without deeper understanding of biology and better research tools. The very
existence of the immune system was unknown. Doctors of the time assumed the
same disease rarely struck the same person twice because a disease "used up"
the materials in the body it needed to live, much the way crops use up soil
nutrients and die unless they are rotated.

In the 19th century, scientists first learned that bacteria cause many
diseases. As the new century began, they discovered even smaller organisms,
viruses, and developed techniques for growing and studying bacteria and
viruses in the laboratory.

In 1906, the Nobel Prize for medicine was awarded to Drs. Camillo Golgi and
Santiago Ramony Cajal, who perfected new chemicals to enhance the visibility
of nerve cells under the microscope. Equipped with this new technology, Dr.
James Dawson at the University of Edinburgh in 1916 performed detailed
microscopic examinations of the brains of patients who had died with MS.

Dr. Dawson wrote a description of the inflammation around blood vessels and
the damage to the myelin with a clarity and thoroughness which has never
been improved. But so little was known about the brain's function that the
meaning of these changes could only be guessed at.

Complexities - and an unrecognized breakthrough

In the decade after World War 1, MS research grew more complex.
Abnormalities in spinal fluid were noted for the first time in 1919, though
their significance was a puzzle. Myelin, which had been discovered in 1878
by Dr. Ranvier, was studied intensively under the microscope and the cell
that makes myelin, the oligodendrocyte, was discovered in 1928.

The first electrical recording of nerve transmission, by Lord Edgar Douglas
Adrian in 1925, established techniques needed to study the activity of
nerves and launched a series of experiments to determine just how the
nervous system works. Ultimately, six Nobel Prizes were awarded for these
studies. The resulting knowledge included clarification of the role of
myelin in nerve conduction and a realization that demyelinated nerves cannot
sustain electrical impulses.

At this time, scientists suspected that some form of toxin or poison caused
MS. Because most MS damage occurs around blood vessels, it seemed reasonable
that a toxin circulating in the bloodstream leaked out into the brain, even
though no researcher could find a trace of it.

Just before World War 11, an important breakthrough occurred. An animal
model of MS was developed out of research on vaccines. It had been known
that people vaccinated against viral illnesses, especially rabies, sometimes
developed a disease resembling MS. It had been assumed that this occurred
because the virus in the vaccines was not completely inactivated. Rabies
vaccines were prepared by growing the rabies virus in nerve tissue.

In 1935, Dr. Thomas Rivers at Rockefeller Institute in New York City
demonstrated that nerve tissue, not viruses, produced the MS-like illness.
By injecting myelin he knew to be virus-free into laboratory animals under
the proper conditions, he could induce their immune systems to attack their
own myelin, producing a disease very similar to MS.

This laboratory animal form of MS, called experimental allergic
encephalomyelitis, or EAE, would later become an important model for
studying the immunology and treatment of MS. In fact, it paved the way to
modern theories of autoimmunity, for it demonstrated how the body can
generate an immunologic attack against itself.

But most doctors in the 1930s were still analyzing toxins or checking blood
circulation in MS. The importance of EAE to NIS was virtually ignored.

Instead, a flurry of experiments in lab animals demonstrated that blocking
the blood supply to the brain sometimes caused myelin to die. The damage
looked a bit like MS. Doctors wondered if MS was caused by circulation
problems and they tried therapies to stimulate blood flow including blood
thinners and drugs to dilate blood vessels. X-rays were also used to treat
NIS, although more for their novelty than for any sound scientific reason.

It would be many years before the essential similarity of EAE and MS was
understood and a link between the immune system and MS was forged.

1940s: The coming of the National MS Society...

World War II focused the energies of the scientific world on new
technologies. New methods and new understandings emerged from wartime
research efforts in many areas. In 1943, for example, the actual composition
of myelin was determined. Then when peace came, one of the most important
catalysts in the fight against NIS was created. The National Multiple
Sclerosis Society was founded in 1946.

Sylvia Lawry, an extraordinary ordinary citizen whose brother suffered from
the disease, placed a classified advertisement in The New York Times asking
to hear from anyone who had recovered from MS. But all the letters she
received came from others who also sought help and hope.

Instead of being discouraged, Ms. Lawry mobilized a group of friends and
advisors, including some who had answered her ad. From this, the National MS
Society was formed to promote contacts among neurologists around the country
who treated MS and to raise money to fund a search for answers.

Reprinted from Summer 1996 issue of Inside MS published by the National
Multiple Sclerosis Society (NMSS)



Top Ten Events...Thus Far

1868: MS described by Jean-Martin Charcot.

1878: Myelin discovered by Louis Ranvier.

1916: Detailed microscopic description made by James Dawson revealed the
basic damage done in MS.

1935: An animal form of NIS (EAE) developed by Thomas Rivers, ultimately
suggesting an autoimmune basis for the disease.

1946: National Multiple Sclerosis Society founded by Sylvia Lawry.

1948: Under an early NMSS grant, oligoclonal bands discovered in the spinal
fluid by Elvin Kabat and others, provided a diagnostic test suggestive of MS
and linking MS to immune system problems.

1965: Definite criteria for MS diagnosis developed by NMSS expert committee.

1969-1970: ACTH used to treat MS exacerbations. This was the first
controlled trial of a successful treatment for MS: it used newly
standardized diagnostic criteria and rating scales to evaluate the efficacy
of treatment.

1981: MRI first used to examine a person with MS. MRI revolutionized
diagnosis and provided evidence that MS is a constantly active disease even
when symptoms abate.

1993: Beta-interferon 1b (Betaseron) approved as the first drug to alter the
course of MS.

Note: Notice that the date at which MS was first described correlates
directly with the start of the industrial age in which chemicals became more
widely used in industry and agriculture in the United States.


--
| The brighter the stupid burns, the more |
| chance that someone will see the light. |
+- D. C. Sessions -+