Tetanus reality and dangers of the tetanus vaccine

There is NO DIAGNOSTIC test for tetanus - they just look at you and
decide because you have a certain set of symptoms that you have
tetanus.
Many things cause similar symptoms - poisonings of many
kinds...............

Tetanus - Hilary Butler on Tetanus


You will see that 'they' (science) don't have the answers, don't have a
clue
many poisoning symptoms resemble tetanus
some bug bite symptoms resemble tetanus

Do you get immunity with tetanus - do you even have tetanus?????

There is NO DIAGNOSTIC test for tetanus - they just look at you and
decide because you have a certain set of symptoms that you have
tetanus.



Here is a long post from Hilary Butler (many of you know her from
mothering.com board)
She has researched vaccines for MANY MANY years and testifies in court
cases and helps defened parents wrongly accused of harming their
children when it is vaccine damage, and helps in cases to try and prove
vaccine damage

Sheri


______________________________________________
By email

Hilary Butler on Tetanus

Tetanus is another one of those things which I spent years researching.
I have a long tome which is not in a format to put it into E, so you
will be spared another Sheri post. Instead you will get a tome of a
different kind. No, I would not have the vaccine, and in making that
choice, this is some of the information I would have thought about. I'm
not putting it all, otherwise I'd be here writing, in a weeks time...

FACTS

The first is that Australia did a study the results of which were
published in the New Zealand Doctor 18, March 1993, pg 23. This was the
first time a study like this had ever been done..., to see what the
antibody levels were in people 10 years after their last booster. Here
is what the medical article said:

quote:

Too many tetanus shots Some people having tetanus boosters already have
antibody levels more than 8,000 times (eight thousand) the protective
level needed, suggesting Australia's regime of a booster every 10 years
is in need of a change. Community physicians say Australia should adopt
the UK regime which advises after five doses, further boosters are
unnecessary.

So the first thing maybe, is go have a titre count done.

But lets look at this titre thing. Way back in the 80's there was a
study done in the Scandinavian J Infect Dis 1983: 15;303 -306, which
looked at soldiers who had been given heaps of tetanus vaccine. Their
antibody levels were way too high as well, and what they discovered was
that "repeated exposure to an antigen, or an overdose of antigen, will
not continually enhance the immune response and may lead to inhibition
or telerance at either T or B cells level.. in addition, a possible
feedback effect stimulated by the high level of toxoid antibodies may
suppress, as Stevens
and Saxon have illustrated, the production of more antibodies at a
later date"

Okay? Do you understand the implications of this?

It came out loud and clear in The Journal of Family Practice, 1997, Vol
44, No 3, pg 299 - 303 when a 29 year old man was admitted with severe
generalised tetanus despite having had a primary series and two booster
injections. And his levels were only 100 times that considered
protective, not 8,000 times as per the Australian study!!!.

Now, if you look at the New Zealand scene, where it was pointed out in
the New Zealand Medical Journal (24th November 1994)that most people
over 35 are unlikely to have had a vaccine, since it was only inroduced
in 1960, then you have to ask yourself why every over-40 year old,
isn't dropping
dead with tetanus. In 1987 a serum survey showed that just over 50% of
the 60-65 group had immunity to tetanus. 83% of the 20-29 year olds,
64% of the 30 -35 year olds. And the stats aren't that much better
today. I understand they are even lower for the USA than for here. So
where did the people with immunity get it from? Many were not
vaccinated...see later...

Now, if you have a look at Tetanus in America, one of the most
interesting articles is a 1969 one from the New England Medical
Journal, Volume 280, Number 11, March 13. And on pages 570 there is a
really interesting decline graph for mortality rates, which shows that
the mortality rate plummetted dramatically from 64/100,000 in 1900to
8/100,000. in 1940. By 1950, with most mothers still unvaccinated, it
was 4.5/100,000.

Not that Tetanus was ever a very common cause of death before hand
anyway, in relation to all the other things that historically could do
you in. Marasmus was a much more common killer of children than
neonatal tetanus ever was... for instance.

They say that it may have been the use of anti-toxin from 1923, but I
know far too much about antitoxin to believe that!!! Antitoxin can kill
all by itself, primarily because it is made in horses, and has
horrendous side-effects in its own right. And some people treated with
anti-tetanus toxoid, will die of the toxoid side-effects, but that is
the risk you take, if you think yu have a chance of dying anyway.....

But the article also recognises that the anti-toxin is not the only
reason, because they cover their bums by also talking about improved
obstetric techniques and neonatal care. And I would have to say wound
care also. But the graph is pretty amazing.

In interesting study in the American Journal of Public Health, August
1984, Vol 74, No 8, showed that in 1,900 adults over 20 years of age,
the overall percentage immunised was 38.6%. Now if, in 1979, in
american, only 38.6% of adults were immunised, what are the factors
which operated then to prevent thousands of adults daily dying from
tetanus?

And obviously there are people still around in USA who still have never
been vaccinated. and haven't died yet. But let's look at something else
as well. As to why tetanus has always been a rare disease in civilian
communities in places like America.

The medical profession has always stated that a person does not acquire
natural immunity to tetanus. But the funny thing is that in 1975 in
Dakar, in the proceeding of the 4th international symposium on tetanus,
they talked about "latent" natural immunity causing reactions to
primary immunisation. Then there was the study in JAMA Nov 19, 1982,
Volume 248, No 19, in which a large number of the unvaccianted Amish
showed serological evidence of immunity to both diphtheria and tetanus.

(Incidentally, the same was though about rabies (that there is no
natural immunity), until a recent study showed that in Alaskan trappers
who had never had any vaccine, they too had antibodies. Poof goes that
theory)

Now, New Zealand should be THE prime hot-bed for tetanus. Everywhere
you turn, you fall over horses, etc etc - cows, sheep dogs. opossums,
you name it. And even here, Tetanus occurs sometimes, but not often.
And what has been most interesting to me is in WHOM it occurs.
Recently, I was able to to a bit of poking into the lives of 4
self-confessed tetanus sufferers who were in the stats. And every
single one of them had factors in their lives which pre-disposed them
to tetanus. One had even had a 3 primary 1 booster schedule, and she
ate refined foods, drank, smoked, and did recreation drugs. Well, hey.
What state was her immune system in!!! didn't stand a
chance at doing its job...

The other three ate the usual white flour, white sugar, coke,
biscuits,very little fruit and vegetables.....

One one was what I would call reasonable healthy, but he smoked like a
train - which as I showed him from the med lit, suppresses the immune
system considerably, especially in the airways and gut area...

Now, someone here said tetanus is only a risk with rusty nails /horses
etc.

It would be wonderful if that were so, but it isn't.

For a start, 5% of us carry it in our guts, happily replicating. In
fact, at one time, the best way to get tetanus in the medical
literature was to have a hemorrhoid dealt with. Until they got wise to
that one..

Tetanus is ubiquitous in our environment. It is everywhere, on
everything.

Where do you think it comes from on the rusty nail? Why do you think it
is that the highest rate of tetanus in vaccinated people is in homeless
drug shooters?

Go get some dust from your mantle-piece tested and it will be there, 10
to the power 6. It is found in bullet wounds, human bite wounds (!!) on
your carrots - you likely eat it every day. There is nowhere that
tetanus is not. In your carpet, on your soil, and often, on your skin.
And if you don't believe me, read the medical literature.

And far from rusty nails being the most common cause of tetanus, that
is
not true. Rusty nails account for less that 40% of tetanus. Most
tetanus
comes where there is no discernable "portal of entry". It has even been
identified following ear-ache (which left me somewhat gob-smacked ---
did
the kid have grommets? and the thing shoved in the ear to "look see",
transfer Tetanus Clostridium? That wasn't even considered...)

Lets look at a bit more history from the medical literature. It has
always been known that war-time historically showed up the highest rate
of tetanus. Far higher than in civilians. Bullet/schrapnel wounds and
all, and the stress of fighting.

Boer war .28 of every thousand wounded got tetanus.
Crimean war 2.0 per 1,000
Am. Civil war 2.0 per thousand
Western front (Flanders horse country WWI average 1.47/thousand
wounded. 2nd world war varied from .06 - .43 per thousand. ( and not
everyone there was vaccinated either. In the paper on the American
Tetanus cases, most who got tetanus had been vaccinated....)

(The difference between the 2nd WW and the first has nothing to do with
the vaccine in my opinion. The second world war was in many ways, far
more hygienically fought. Where were all the mud-filled trenches, and
ghastly living conditions in the second world war, compared to the
first? Those who
know their history will see that right away...)

Now, the question should be, not why did the few actually GET tetanus,
but why didn't the other 998 per thousand wounded get it? Especially
when you realise that it was days, if not weeks, before those wounded
people got any serious attention. When you read about weeks in stinking
ships amongst the wounded from Gallipoli on their way to hospital in
EGYPT as well as the substandard treatment, you have to wonder why they
all didn't die.

The answer lies IMO in the unique susceptibility of the individual.
That a vaccine can confer immunity is not the question that needs to be
answered. The medical profession attributes the decrease in Tetanus
SOLELY to vaccination which is grossly misleading.

I mean, look at your family trees? Did all your great-grandparents die
of tetanus? Nope. Did they never get exposed to it? ACtually, they had
far more wounds likely to have harboured it, than we do today...

A good read of the medical textbooks of the 20's gives some idea as to
one chief reason..... In a study done in 1919-1920 by Dr Harriette
Chick of the lister Institute, and other working in Pirquets clinic in
Vienna, showed that "nearly all children in Central Europe were more or
less rachitic"

Remember Rickets? Remember being lined up with cod-liver oil and
black-strap molasses. (Last year, there was an article which shows that
rickets is rising again in the USA....)

Most older people living now can remember the free milk, cod-liver oil
by the spoon-full. One here has already made derogatory comments about
it.... Even in the rich communities in London in the 30's there were
still over 30% of children with definable nutritional deficiencies...

Now, its interesting in that light, to know that there are studies
which show that when you vaccinate people whose nutritional status is
not good, at least 30% do not respond to the vaccine, even when given
highly concentrated fluid toxoids (Med J Aust, Aug 7, 1976). Again the
work by Chandra shows that their rate of non-conversion to the vaccine
gets higher with the degree of malnutrition, and that malnourished
mothers pass on such low levels of IgG that the babies' passive
immunity is virtually nil.

There is no way around the blinding revelation of the obvious that

1) Bad nutrition is a very serious immune suppressor

2) Immune suppression, or malfunction, is the chief determiner of the
severity of tetanus.

And the work by Chandra et all, shows that obesity causes immune
changes similar to those caused by malnutrition, so nutritional status
is wide-ranging in its implications.

And then there is the hysteria factor which suppresses the immune
system as well, which was amply demonstrated in the USA in Johb Paul's
book "The history of Polio" where at times when the media hype was
bordering on the screaming paranoid, people would be admitted with what
they called
"pseudo-polio"

Talking of hysteria, rember that hysteria is one of the differential
diagnoses of tetanus, as is hypocalcemia, meningitis, rabies, drug
withdrawal, strychnine poisoning, and dystonic drug reaction.

"red streaks" may have nothing to do with tetanus at all. When I was in
USA in 1993, I got a chigger bite, which the chemist treated with what
looked like nail polish, and said it would be just fine. Well, it
wasn't. After 4 days, my legs ached, and I had red streaks up to the
groin. And while my hosts freaked their heads off, I plonked myself in
the bath, took a sterilised 3 inch needle, put a piece of wood between
my teeth, and opened it up in a Y shape. The rotten chigger, and pus
exploded everywhere....

And within three days, the red streaks had completely gone as had all
infection. And since I am fatally allergic to all antibiotics, no, I
didn't use them. I let my body do the job it is designed for, with
judicious help from vitamin quackery.....

Back to history

There is much made of the fact that the Japanese soldiers in WWII had a
lot of tetanus in comparison to the Americans. 14 per hundred.... But
when researching this, I was interested to read this "We embarked 284
wounded Japanese soldiers. We found them to be generally dirty,
emaciated and with wounds that had been improperly treated." By
contrast, the 384 wounded US
solders were described as clean, well-fed with wounds that had been
promptly treated.

Aparently this counted for nothing, and that the reason the USA
soldiers had escaped "in comparison" was the vaccine. Which makes me
very annoyed, because there is another factor they missed out, which is
integral to understanding the japanese-- What was not taken into
account was that to be taken captive was the ultimate in disgrace to
any japanese, and many lost the will to live, and died anyway. Imagine
the shape of the immune system of someone with no hope....

Now you would think too, that ALL the US personnel during WWII who got
tetanus would have been unimmunised, but not so. There were twelve
cases. 6 of them had completed an immunisation schedule, and one had
had one shot. And three of the 6 with a completed schedule, died. Three
of the unvaccinated suvived. The one who had had one shot, survived.
Seems sort of 50/50 to me. (Bull US Army Med Dept, Vol VII, no 4, April
1947). The British records are even stranger, with the immunized often
having a higher overall case mortality record than the unimmunsed. In
the Lancet, Jan 26, 1946, pg 116 looking at the case Mortality rates,
in the "protected" it was 50%, in the Unprotected it was 46.8%, and in
the incomplete or doubtful it was 42.1%.

The list of side-effects which I have for Tetanus vaccine is huge, but
I won't go into that. What I wanted to reinforce was nutritional
status, and some history, but finally to draw your attention to a most
interesting study. This is found in the Bangladesh medical Research
Council Bulleting, Volume 10, No 1, June 1984.

As I said, a differential diagnosis is Strynchnine poisoning. The first
part was to take two day old chicks divided into fur groups with 15
birds in each. Group one got 5 nanograms of strychnine sulphate (SS).
Group 2 got SS plus 30 mg of Ascorbic Acid (Vitamin C) Group 3 10
nonograms of SS, and group 4 got10 nanos SS and 30 mg Vitamin C. The
results we

Group one, Wings of all birds stretched, Some walked on toes, others
kept jumping but could not walk.

Group 2 - No symptoms

Group 3, Extensor paralysis of legs, opisthotonus and severe
convulsions,and all but three died.

Group 4, Extensor paralysis in 3 chicks. No neurological symptoms in
others. the affected bird recovered in about 30 minutes after the
appearance of symptoms.

Then they randomly assigned tetanus cases into four groups.

Ages 1-11

No Vitamin C = 72% mortality rate.

Vitamin C 0% mortality rate.

Ages 13 - 30

Vitamin C, 37% mortality

Non Vitamin C 67.8 % mortality rate.

But they made one mistake with this. All patients got 1,000 mg of
Vitamin C. They did not ADJUST UPWARDS the dose of vitamin C for the
bigger body weight. Had they done that, the results might have been
different. They did another study looking at the use of B 6, pyrodoxine
and found that that also reduced both severity an mortality.

Think on that. Tetanus is a toxin-mediated disease, rather like
diphtheria. It is determined by nutritional status, and immune function
more than anything else...

But there is another interesting fact. The body, with a wound, puts up
"road-blocks" to stop the toxin spreading. It is fascinating to me,
that it has long been known that "Wound excision should be delayed
until the antitoxin is given, as free tetanospasmin is mobilized into
the blood-stream during surgical manipulation." In other words,
debridement of wounds breaks open these road-blocks, and floods the
system with poison....

I also found in interesting that in the Am J Dis Child Vol 135, June
1981, Pg 571 it says "the mortality in reported cases of tetanus is
higher in the USA than in developing countries." Perhaps it is because
in the USA the methods of treatment are very invastive, include
debridement, tracheotomy - a whole host of things which just can't be
done in developing countries, not just because of cost, but also
because of infection risk.