Why Did "Aids Baby" Eliza Jane Really Die?

JanD wrote:
"Robert" wrote in message
...


The child was never prescribed that antibiotic before so it is impossible
for that child to develop an anaphylactic reaction on first exposure.
Those
are the facts.

NO, it is NOT impossible.


But highly improbable. Much less probable, in fact, than the idea that
her untreated AIDS had led to her development of PCP, and that said PCP
led to her hypoxic and ultimately fatal state.

But you're the one who apparently thinks it's unfair to label Yurko a
child-killer (despite his manslaughter conviction), so who's to say
exactly what is going on in your shriveled little brain? That a
veterinarian is a pathologist (a title, BTW, only bestowed on medical
doctors who have completed training in human pathology)? That the
Asian liver fluke is responsible for all cancers? That the Jews on
this n.g. are all liars? That all Catholics are going to hell?

Who knows?

Mark, MD

Bonus points if you can explain why it isn't impossible, but is highly
improbable.

Don't forget to answer now

C'mon, blind us with your knowledge and clinical acumen, dear
heart...or are you just arguing for argument's sake?

Mark, MD

On Wed, 07 Dec 2005 03:42:06 GMT, wrote:

On Tue, 6 Dec 2005 13:30:16 -0800, "Robert" wrote:

The child was never prescribed that antibiotic before so it is impossible
for that child to develop an anaphylactic reaction on first exposure. Those
are the facts.


The fact is that the first time she took the antibiotic she had no reaction.
The reaction occurred when she took it again and she got worse as she kept
taking it.


www.aras.ab.ca/EJ/Al-BayatiReport.pdf

Section V. Clinical Indicators of Acute Allergic Reaction to Amoxicillin
Observed in Eliza Jane’s Case.
Eliza Jane received the first dose of amoxicillin (400 mg twice a day) at about
18:25 on
May 14, 2005. Her condition appeared to worsen after receiving the second dose
of
amoxicillin. Her mother described Eliza Jane’s condition during the course of
her
treatment with amoxicillin as follows: On May 15th, Eliza Jane vomited several
times
throughout the day and was pale. After dinner, she seemed agitated and then, at
some
point, lethargic. She had a fever of 101.1-degrees F, but her extremities felt
cold to the
touch [1, 2]. She suffered from cardiac arrest shortly after receiving the
fourth dose on
the evening of May 15th.
Eliza Jane’s acute symptoms developed following receiving the amoxicillin and
the other
clinical observations and biomarkers described in her case indicate clearly that
she died
from acute allergic reaction to amoxicillin. Amoxicillin and other penicillin
type
antibiotics have known to cause serious and fatal allergic reactions in children
and adults.
Amoxicillin has been known to induce anemia, bone marrow depression, and Type I,
II,
III and IV allergic reactions in children and adults [38-62]. The clinical
evidence and the
rapid development of Eliza Jane’s anemia indicate that her anemia and bone
marrow
depression was caused by the synergistic actions of amoxicillin and a viral
infection,
most likely HPV B19.
Below is a list of clinical observations and biomarkers that show Eliza Jane
died from
acute allergic to amoxicillin:
1) The weight of Eliza Jane’s lungs was 306 g and the expected average normal
weight
for her age is 166 g (Table 5). The microscopic examination of the lung sections
did not
reveal inflammation of the lungs and that the excess in lung weight (140 g)
resulted from
edema. The clinical data collected in this case indicate that the fluid observed
in the lungs
developed following treatment with amoxicillin on May 14th. These include:
a) Physicians examined Eliza Jane on May 14th and prior visits and found her
lungs clear.
b) Two chest x-rays were taken at VPH on May 16th. The first x-ray was taken at
0039 and showed small patchy infiltrates. The second x-ray was taken at about

Dr. Al-Bayati’s report, October 25, 2005
Eliza Jane Scovill
34
0500 and showed more diffuse pulmonary edema pattern [2]. These observations
indicate that some fluid moved from the blood vessels into the lungs during
Eliza
Jane’s hospitalization at VPH.
2) Accumulation of fluid also observed in the pleural and peritoneal cavities
and
pericardial sac which indicates cardiovascular problems. Eliza Jane’s pleural
cavities
contained approximately 20 cc of clear serous fluid. In addition a 60 cc of
serous fluid
was collected from the peritoneal cavity. No adhesions or inflammation were
observed in
the in the pleural and peritoneal cavities and pericardial sac. These
observations indicate
that the excess fluid in these compartments resulted from cardiovascular
problems.
Allergic reaction to amoxicillin causes the release of many vasoactive mediators
such as
histamine, which induces vasodilatation and leakage of fluid outside the blood
vessels
[54-56].

"Mark" wrote in message
ups.com...
JanD wrote:
"Robert" wrote in message
...


The child was never prescribed that antibiotic before so it is
impossible
for that child to develop an anaphylactic reaction on first exposure.
Those
are the facts.

NO, it is NOT impossible.


But highly improbable. Much less probable, in fact, than the idea that
her untreated AIDS had led to her development of PCP, and that said PCP
led to her hypoxic and ultimately fatal state.

It is NOT impossible.


http://parentcenter.babycenter.com/r...ies/68016.html

Anaphylactic shock (also called anaphylaxis) is a rapid and severe allergic
reaction. One of the scariest health emergencies a parent can face,
anaphylaxis starts when the immune system mistakenly responds to a harmless
substance as if it were a serious threat, triggering the release of
histamine and other chemicals that cause symptoms - some of them
life-threatening - including:
.. Swelling of the skin, lips, throat, tongue, or face

.. Wheezing or severe breathing problems

.. Rapid pulse or irregular heartbeat

.. Hives

.. Dizziness, fainting, loss of consciousness

.. Nausea, vomiting, abdominal cramps, diarrhea

.. Extremely pale skin and sweating

Symptoms usually appear within minutes or up to two hours after exposure to
the substance, though it's possible for symptoms to show up four hours
later.

.. Drugs in the penicillin family (including the popular antibiotic
amoxicillin)

http://www.allergyclinic.co.nz/guides/12.html

.. Immunosuppression may enhance the sensitising potential of some drugs. An
immunosupressed patient may become deficient in those suppressor T cells
that regulate IgE antibody synthesis.

In recent years, immunosuppression associated with HIV infections has become
a major risk factor for adverse drug reactions. Over half of the patients
with AIDS develop adverse reactions when treated with
trimethoprim-sulfamethoxazole. The incidence of reactions to Ampicillin is
inversely proportional to CD4+ cell counts. Several other drugs have been
found to have a higher than expected tendency to produce adverse reactions
in HIV-infected patients. Most are mild-to-moderate skin eruptions, but the
risk of anaphylaxis and even toxic epidermal necrolysis may also be enhanced
in HIV infection

Most anaphylactic reaction occurs within one hour after the patient takes
the drug



But you're the one who apparently thinks it's unfair to label Yurko a
child-killer (despite his manslaughter conviction),

You need to take a good look at ALL those who have doubts about the case.

http://www.freeyurko.bizland.com/appeal.html#support

I have NEVER been one to go with the flow and shout OFF WITH HIS HEAD!

There are MANY MANY innocent people behind bars because of this attitude.

[all diversions + insults snipped]

Sad to say, you just proved yourself to be a liar.

You are NOT here becuase you are interested in childrens health.

You are here to insult and harass.

Mark, MD

"Mark" wrote in message
oups.com...
Bonus points if you can explain why it isn't impossible, but is highly
improbable.

Don't forget to answer now

LOL, I already did, before I read this post.

C'mon with the cheers and the bonus points *;*

C'mon, blind us with your knowledge and clinical acumen, dear
heart...or are you just arguing for argument's sake?

Mark, MD

Thank you, so all of this time, the *gang* has NOT known the facts.

Yet, they claimed they did.

wrote in message
...
On Wed, 07 Dec 2005 03:42:06 GMT, wrote:

On Tue, 6 Dec 2005 13:30:16 -0800, "Robert"
wrote:

The child was never prescribed that antibiotic before so it is impossible
for that child to develop an anaphylactic reaction on first exposure.
Those
are the facts.


The fact is that the first time she took the antibiotic she had no
reaction.
The reaction occurred when she took it again and she got worse as she kept
taking it.


www.aras.ab.ca/EJ/Al-BayatiReport.pdf

Section V. Clinical Indicators of Acute Allergic Reaction to Amoxicillin
Observed in Eliza Jane's Case.
Eliza Jane received the first dose of amoxicillin (400 mg twice a day) at
about
18:25 on
May 14, 2005. Her condition appeared to worsen after receiving the second
dose
of
amoxicillin. Her mother described Eliza Jane's condition during the course
of
her
treatment with amoxicillin as follows: On May 15th, Eliza Jane vomited
several
times
throughout the day and was pale. After dinner, she seemed agitated and
then, at
some
point, lethargic. She had a fever of 101.1-degrees F, but her extremities
felt
cold to the
touch [1, 2]. She suffered from cardiac arrest shortly after receiving the
fourth dose on
the evening of May 15th.
Eliza Jane's acute symptoms developed following receiving the amoxicillin
and
the other
clinical observations and biomarkers described in her case indicate
clearly that
she died
from acute allergic reaction to amoxicillin. Amoxicillin and other
penicillin
type
antibiotics have known to cause serious and fatal allergic reactions in
children
and adults.
Amoxicillin has been known to induce anemia, bone marrow depression, and
Type I,
II,
III and IV allergic reactions in children and adults [38-62]. The clinical
evidence and the
rapid development of Eliza Jane's anemia indicate that her anemia and bone
marrow
depression was caused by the synergistic actions of amoxicillin and a
viral
infection,
most likely HPV B19.
Below is a list of clinical observations and biomarkers that show Eliza
Jane
died from
acute allergic to amoxicillin:
1) The weight of Eliza Jane's lungs was 306 g and the expected average
normal
weight
for her age is 166 g (Table 5). The microscopic examination of the lung
sections
did not
reveal inflammation of the lungs and that the excess in lung weight (140
g)
resulted from
edema. The clinical data collected in this case indicate that the fluid
observed
in the lungs
developed following treatment with amoxicillin on May 14th. These include:
a) Physicians examined Eliza Jane on May 14th and prior visits and found
her
lungs clear.
b) Two chest x-rays were taken at VPH on May 16th. The first x-ray was
taken at
0039 and showed small patchy infiltrates. The second x-ray was taken at
about

Dr. Al-Bayati's report, October 25, 2005
Eliza Jane Scovill
34
0500 and showed more diffuse pulmonary edema pattern [2]. These
observations
indicate that some fluid moved from the blood vessels into the lungs
during
Eliza
Jane's hospitalization at VPH.
2) Accumulation of fluid also observed in the pleural and peritoneal
cavities
and
pericardial sac which indicates cardiovascular problems. Eliza Jane's
pleural
cavities
contained approximately 20 cc of clear serous fluid. In addition a 60 cc
of
serous fluid
was collected from the peritoneal cavity. No adhesions or inflammation
were
observed in
the in the pleural and peritoneal cavities and pericardial sac. These
observations indicate
that the excess fluid in these compartments resulted from cardiovascular
problems.
Allergic reaction to amoxicillin causes the release of many vasoactive
mediators
such as
histamine, which induces vasodilatation and leakage of fluid outside the
blood
vessels
[54-56].

JanD wrote:
"Mark" wrote in message
ups.com...
JanD wrote:
"Robert" wrote in message
...


The child was never prescribed that antibiotic before so it is
impossible
for that child to develop an anaphylactic reaction on first exposure.
Those
are the facts.

NO, it is NOT impossible.


But highly improbable. Much less probable, in fact, than the idea that
her untreated AIDS had led to her development of PCP, and that said PCP
led to her hypoxic and ultimately fatal state.

It is NOT impossible.


http://parentcenter.babycenter.com/r...ies/68016.html

Anaphylactic shock (also called anaphylaxis) is a rapid and severe allergic
reaction. One of the scariest health emergencies a parent can face,
anaphylaxis starts when the immune system mistakenly responds to a harmless
substance as if it were a serious threat, triggering the release of
histamine and other chemicals that cause symptoms - some of them
life-threatening - including:
. Swelling of the skin, lips, throat, tongue, or face

. Wheezing or severe breathing problems

. Rapid pulse or irregular heartbeat

. Hives

. Dizziness, fainting, loss of consciousness

. Nausea, vomiting, abdominal cramps, diarrhea

. Extremely pale skin and sweating

Symptoms usually appear within minutes or up to two hours after exposure to
the substance, though it's possible for symptoms to show up four hours
later.

. Drugs in the penicillin family (including the popular antibiotic
amoxicillin)

http://www.allergyclinic.co.nz/guides/12.html

. Immunosuppression may enhance the sensitising potential of some drugs. An
immunosupressed patient may become deficient in those suppressor T cells
that regulate IgE antibody synthesis.

In recent years, immunosuppression associated with HIV infections has become
a major risk factor for adverse drug reactions. Over half of the patients
with AIDS develop adverse reactions when treated with
trimethoprim-sulfamethoxazole. The incidence of reactions to Ampicillin is
inversely proportional to CD4+ cell counts. Several other drugs have been
found to have a higher than expected tendency to produce adverse reactions
in HIV-infected patients. Most are mild-to-moderate skin eruptions, but the
risk of anaphylaxis and even toxic epidermal necrolysis may also be enhanced
in HIV infection

Most anaphylactic reaction occurs within one hour after the patient takes
the drug

So you're saying the child did have AIDS?





But you're the one who apparently thinks it's unfair to label Yurko a
child-killer (despite his manslaughter conviction),

You need to take a good look at ALL those who have doubts about the case.

http://www.freeyurko.bizland.com/appeal.html#support

I have NEVER been one to go with the flow and shout OFF WITH HIS HEAD!

There are MANY MANY innocent people behind bars because of this attitude.

[all diversions + insults snipped]

Sad to say, you just proved yourself to be a liar.

You are NOT here becuase you are interested in childrens health.

You are here to insult and harass.

Mark, MD

JanD wrote:
"Mark" wrote in message
oups.com...
Bonus points if you can explain why it isn't impossible, but is highly
improbable.

Don't forget to answer now

LOL, I already did, before I read this post.

Sure you did, Jan.


C'mon with the cheers and the bonus points *;*

C'mon, blind us with your knowledge and clinical acumen, dear
heart...or are you just arguing for argument's sake?

Mark, MD

JanD wrote:
Thank you, so all of this time, the *gang* has NOT known the facts.

Yet, they claimed they did.

Bless. Yet again you've obviously failed to read the posts and links
you comment on.
Allow me to direct you, as others already have, to the link provided by
HCN a couple of days ago, in which Nick Bennett addresses the issues
brought up by al-Bayati, and demonstrates al-Bayati's lack of
understanding of the field in which he was egregiously commenting:

http://catallarchy.net/blog/wp-conte...t_on_Eliza.pdf

Perhaps you could attempt a rebuttal of some of Bennett's points, Jan?
Instead of wrongly claiming that no-one knew what al-Bayati said in the
first place.

LOL.

Cathy


wrote in message
...
On Wed, 07 Dec 2005 03:42:06 GMT, wrote:

On Tue, 6 Dec 2005 13:30:16 -0800, "Robert"
wrote:

The child was never prescribed that antibiotic before so it is impossible
for that child to develop an anaphylactic reaction on first exposure.
Those
are the facts.


The fact is that the first time she took the antibiotic she had no
reaction.
The reaction occurred when she took it again and she got worse as she kept
taking it.


www.aras.ab.ca/EJ/Al-BayatiReport.pdf

Section V. Clinical Indicators of Acute Allergic Reaction to Amoxicillin
Observed in Eliza Jane's Case.
Eliza Jane received the first dose of amoxicillin (400 mg twice a day) at
about
18:25 on
May 14, 2005. Her condition appeared to worsen after receiving the second
dose
of
amoxicillin. Her mother described Eliza Jane's condition during the course
of
her
treatment with amoxicillin as follows: On May 15th, Eliza Jane vomited
several
times
throughout the day and was pale. After dinner, she seemed agitated and
then, at
some
point, lethargic. She had a fever of 101.1-degrees F, but her extremities
felt
cold to the
touch [1, 2]. She suffered from cardiac arrest shortly after receiving the
fourth dose on
the evening of May 15th.
Eliza Jane's acute symptoms developed following receiving the amoxicillin
and
the other
clinical observations and biomarkers described in her case indicate
clearly that
she died
from acute allergic reaction to amoxicillin. Amoxicillin and other
penicillin
type
antibiotics have known to cause serious and fatal allergic reactions in
children
and adults.
Amoxicillin has been known to induce anemia, bone marrow depression, and
Type I,
II,
III and IV allergic reactions in children and adults [38-62]. The clinical
evidence and the
rapid development of Eliza Jane's anemia indicate that her anemia and bone
marrow
depression was caused by the synergistic actions of amoxicillin and a
viral
infection,
most likely HPV B19.
Below is a list of clinical observations and biomarkers that show Eliza
Jane
died from
acute allergic to amoxicillin:
1) The weight of Eliza Jane's lungs was 306 g and the expected average
normal
weight
for her age is 166 g (Table 5). The microscopic examination of the lung
sections
did not
reveal inflammation of the lungs and that the excess in lung weight (140
g)
resulted from
edema. The clinical data collected in this case indicate that the fluid
observed
in the lungs
developed following treatment with amoxicillin on May 14th. These include:
a) Physicians examined Eliza Jane on May 14th and prior visits and found
her
lungs clear.
b) Two chest x-rays were taken at VPH on May 16th. The first x-ray was
taken at
0039 and showed small patchy infiltrates. The second x-ray was taken at
about

Dr. Al-Bayati's report, October 25, 2005
Eliza Jane Scovill
34
0500 and showed more diffuse pulmonary edema pattern [2]. These
observations
indicate that some fluid moved from the blood vessels into the lungs
during
Eliza
Jane's hospitalization at VPH.
2) Accumulation of fluid also observed in the pleural and peritoneal
cavities
and
pericardial sac which indicates cardiovascular problems. Eliza Jane's
pleural
cavities
contained approximately 20 cc of clear serous fluid. In addition a 60 cc
of
serous fluid
was collected from the peritoneal cavity. No adhesions or inflammation
were
observed in
the in the pleural and peritoneal cavities and pericardial sac. These
observations indicate
that the excess fluid in these compartments resulted from cardiovascular
problems.
Allergic reaction to amoxicillin causes the release of many vasoactive
mediators
such as
histamine, which induces vasodilatation and leakage of fluid outside the
blood
vessels
[54-56].

wrote in message
...
Eliza Jane received the first dose of amoxicillin (400 mg twice a day) at
about
18:25 on
May 14, 2005. Her condition appeared to worsen after receiving the second
dose
of
amoxicillin. Her mother described Eliza Jane's condition during the course
of
her
treatment with amoxicillin as follows: On May 15th, Eliza Jane vomited
several
times
throughout the day and was pale. After dinner, she seemed agitated and
then, at
some
point, lethargic. She had a fever of 101.1-degrees F, but her extremities
felt
cold to the
touch [1, 2]. She suffered from cardiac arrest shortly after receiving the
fourth dose on
the evening of May 15th.

24 hours is not immediate and not typical of a Type I IgE type of reactions.
It takes time to develop the total amount of antibody needed to get the
massive response. Mast cells throughout the body would have to be covered
with this specific IgE to the drug and it would be highly impractical and
practically unheard of for this to happen from first exposure to anaphylaxis
within 24 hours.
The child was pale (blue?) before she was given the antibiotic.
That's about all I can trust. I would rather see the coroner report if you
don't mind and not somebody's interpretation of what he read. I would like
to see the findings by the person who did the autopsy.