Elimination of mercury

"Peter Bowditch" is eager to post Mark Probert's lies.

Then posts he own. Then SPAM SPAM SPAM.
wrote:


Mark Probert wrote:

Clearly, the claim by the Mercury Militia that it accumulates after each
vaccination is not supported by this research. Ethyl Mercury, the
byproduct of thimerosal metabolism is eliminated rapidly, and is gone
before the next vaccination.

Mark clearly lied..............REPEATEDLY.

Mark also refused to discuss the vested interest of the CDC and FDA
in vaccines. He also did not watch the video.

Blood work from kids SHOULD have been taken within two to four hours, NOT
days
after vaccines, thimerosal crosses the blood brain barrier and is stored in
the
brain.


Kids were given as much as seven to nine shots a day and thimerosal level
reaching 100% OVER the acceptable limit.


Rogam (sp?) was given to pregnant women, doctors not knowing it contained
thimerosal.


At a conservative rate of 10% of kids affected, that's 50,000 kids in the
US.
Rate likely to be higher.


The CDC says records of adverse effects can be found, however it was told
the
hoops people must jump through to get these records. One doctor even
submitted
150 pages to get through this red tape, but that was not good enough.


Invalid data in reaching the statistics of affected kids.
Pediatrics. 2001 May;107(5):1147-54.



Thimerosal neurotoxicity and protection with
N-Acetylcysteine supplementation", Health Sentinel, January 3, 2005,

In the 1930s, Eli Lily developed Thimerosal as a preservative and it
was widely used in vaccines. Until the removal of Thimerosal, which
contains 49.9% ethyl mercury by weight, from most pediatric vaccines in
2001, the source of the largest human exposure to mercury in the US was
in children under 18 months of age undergoing routine childhood
immunization schedules. Before 2001, a child may have received a
cumulative dose of over 200 µg/kg (micrograms per kilogram) in the
first 18 months of life.

Although Thimerosal has been removed from most childhood vaccines, it
is still present in the flu vaccine, which is given to pregnant women,
the elderly, and children. Also, many vaccines given to children in
developing countries still contain Thimerosal.

In the 2005 issue of NeuroToxicology, the authors of a study examine
the toxicity of Thimerosal within the body including neurons. They
examine the neurotoxic mechanisms, how the body detoxifies mercury, and
the use of N-Acetylcysteine, or NAC for short, in facilitating the
detoxification pathway within the body.

Glutathione, a tripeptide composed of cysteine, glutamate, and glycine,
is manufactured in the liver and also in the brain. Normally, the
concentrations of glutathione in the cells are quite high providing for
detoxification of a variety of heavy metals including mercury. However,
when this essential antioxidant is depleted the excess mercury can bind
to internal cellular proteins leading to toxic damage. Studies have
shown that, "low micromolar concentrations of Thimerosal induced DNA
strand breaks, caspase-3 activation, membrane damage and cell death."

Although the brain can produce glutathione, it can only manufacture
this from its immediate precursor cysteine. The liver, on the other
hand, is able through a long series of biochemical steps to create
glutathione from methionine. Methionine is an essential amino acid that
supplies the body with sulfur and methyl groups. The liver uses a
number of enzyme systems along with various B vitamins to produce
glutathione. The liver then exports the glutathione to the blood that
then is broken down to cystine. Cystine crosses the blood-brain barrier
to be used by the brain to make glutathione. Thus, the brain is reliant
on the liver to manufacture chemicals to keep it free from toxins.

The brain contains neurons and other cells called astrocytes.
Astrocytes use the cystine that crosses the blood-brain barrier to make
glutathione. The astrocytes then export the glutathione to the space
between the cells where it is broken down to cysteine. The neurons take
up the cysteine and manufacture glutathione. This complex series of
biochemical events is what is necessary to keep the brain free from
heavy metal damage.

The authors first examined the level of Thimerosal that would cause
toxic damage to cells. They found that the higher the concentration of
Thimerosal the greater the number of cells that were killed although
the nerve cell response occurred with only a 3 hour exposure, whereas
the other cell line required a 48 hour exposure demonstrating that
nerve cells are more sensitive to Thimerosal toxicity. "In both cell
lines, a progressive increase in cytotoxicity (decrease in viability)
was observed when Thimerosal dose was progressively doubled from 2.5
µmol/L [micromoles per liter] to 5, 10, and 20 µmol/L. Viability was
reduced more than 50% in both cell lines with exposure to 10 µmol/L
Thimerosal and less than 10% of cells survived a dose of 20 µmol/L."

The authors then pretreated cells with NAC before adding a dose of 15
µmol/L Thimerosal. They found that, "Thimerosal alone induced more
than a 6-fold decrease in viability", and that NAC, "provided
significant protection against cell death". The authors note,
"Thimerosal induces oxidative stress and apoptosis by activating
mitochondrial cell death pathways. A subsequent study using cultured
human neuron and fibroblast cell lines similarly showed that low
micromolar concentrations of Thimerosal induced DNA strand breaks,
caspase-3 activation, membrane damage and cell death."

The authors conclude that, "numerous clinical studies have
demonstrated the efficacy of NAC in increasing intracellular
glutathione levels and reducing oxidative stress in humans. Since
cytotoxicity with both ethyl- and methyl- mercury have been shown to be
mediated by glutathione depletion, dietary supplements that increase
intracellular glutathione could be envisioned as an effective
intervention to reduce previous or anticipated exposure to mercury.
This approach would be especially valuable in the elderly and in
pregnant women receiving Rho D immunoglobulin shots, and individuals
who regularly consume mercury-containing fish."

SOURCE: NeuroToxicology, Vol. 26, 2005, pp. 1-8


Shhhhhhh, your gonna ruin a perfectly good conspiracy. Next thing you
know they will be blaming it on sunspots, florination of the water, or
inorganic farming.

Eric

snip more personally trashing
--
Peter Bowditch

"john" wrote in message
...

"Peter Bowditch" wrote in message
...

Joe Mercola has a page blaming fluoridation for autism. Of course, he
also has another page blaming mercury and yet another blaming, wait
for it, pasteurised milk. But what else would you expect from him -
consistency?
--

You consistently state the same bull**** --that autism couldn't be caused
by more than one thing

Speaking of consist things he says.

There is no such thing a mercury amalgam. Medline disagrees with him.

From IIean. Thank You.


http://groups.google.com/group/misc....browse_frm/thr...


I went to Medline ... there were 235 studies using that term ... the
first 20 I've copied below.



http://search.medscape.com/uslclient/searchMedline.do?queryText="mercury%20amalgam"


MEDLINE Results Results 1 - 20 of 235
1 2 3 4 5 6 7 8 9 10 next»


Effect of restoration of children's teeth with mercury amalgam on the
prevalence of mercury- and antibiotic-resistant oral bacteria.The
purpose of this study was to determine whether placement of mercury
amalgam restorations in children's teeth induces an increase in oral
bacteria resistant to mercury, penicillin, ampicillin, erythromycin,
or tetracycline.
from Microb Drug Resist - Jan 2003 - Rachel Pike, Victoria Lucas,
Aviva Petrie, et. al.


Mercury-reactive lymphocytes in peripheral blood are not a marker for
dental amalgam associated disease.OBJECTIVES: The popular press and
publications associated with alternative medicine increasingly report
that chronic ill health, particularly myalgic encephalitis like
conditions, are associated with mercury amalgam fillings.
from J Dent - Sep 2001 - D C Henderson, R Clifford


Scanning electrochemical microscopy imaging of rhodochrosite
dissolution using gold amalgam microelectrodes.Gold/mercury amalgam
(Au/Hg) microelectrodes with a diameter of 25 microm were developed
for the detection of environmentally relevant analytes such as
manganese and iron by scanning electrochemical microscopy (SECM), and
applied to investigate the controlled dissolution of manganese
carbonate (MnCO(3); rhodochrosite) in acidic conditions.
from Analyst - May 2004 - Douglas Rudolph, Stephanie Neuhuber,
Christine Kranz, et. al.


Prevalence and antibiotic resistance profile of mercury-resistant oral
bacteria from children with and without mercury amalgam fillings.Genes
encoding resistance to mercury and to antibiotics are often carried on
the same mobile genetic element and so it is possible that
mercury-containing dental materials may select for bacteria resistant
to mercury and to antibiotics.
from J Antimicrob Chemother - May 2002 - R Pike, V Lucas, P
Stapleton, et. al.


The relationship between amalgam restorations and mercury levels in
male dentists and nondental health professionals.OBJECTIVES: The
objectives of this study we (1) to compare the mercury levels in
general dentists with the mercury levels in other health professionals
using toenail clippings as a biomarker, (2) to identify risk factors
associated with high mercury levels, and (3) to compare practice
characteristics of dentists with high and low mercury levels.
from J Public Health Dent - Jan 2003 - Anil Joshi, Chester W
Douglass, Hyun-Duck Kim, et. al.


Syntheses, structure, and derivatization of potassium complexes of
penta(organo)[60]fullerene-monoanion, -dianion, and -trianion into
hepta- and octa(organo)fullerenes.Two-electron reduction of
penta(organo)[60]fullerenes C(60)Ar(5)H (Ar = Ph and biphenyl) by
potassium/mercury amalgam afforded potassium complexes of the
corresponding open-shell radical dianions [K+(thf)n]2[C60Ar5(2-.
from J Am Chem Soc - Jun 2005 - Yutaka Matsuo


Urinary mercury concentrations associated with dental restorations in
adult women aged 16-49 years: United States, 1999-2000.BACKGROUND:
Mercury amalgam dental restorations have been used by dentists since
the mid 19th century and issues on safety continue to be periodically
debated within the scientific and public health communities.
from Occup Environ Med - Jun 2005 - B A Dye, S E Schober, C F
Dillon, et. al.


Determination of glutathione in single human hepatocarcinoma cells by
capillary electrophoresis with electrochemical detection.A method for
determination of glutathione (GSH) in single human hepatocarcinoma
(HH) cells was described by capillary zone electrophoresis with
electrochemical detection at a gold/mercury amalgam micro-disk
electrode.
from J Chromatogr B Analyt Technol Biomed Life Sci - Jun 2003 - Wei
Wang, Hua Xin, Honglian Shao


An uncertain risk and an uncertain futu assessing the legal
implications of mercury amalgam fillings.No abstract available
from Health Matrix Clevel - Jan 2004 - Mary Ann Chirba-Martin


Multiple sclerosis and dental amalgam: case-control study in Ferrara,
Italy.Dental amalgam fillings containing mercury have been suggested
as a possible risk factor for multiple sclerosis (MS).
from Neuroepidemiology - May 2001 - I Casetta, M Invernizzi


Dental amalgam and selenium in blood.It has been suggested that
selenium (Se) exhibits protective effects against mercury (Hg)
toxicity in humans due to formation of a Hg-Se complex bound to
selenoprotein P in blood.
from Environ Res - Dec 2001 - P J Høl, J S Vamnes, N R Gjerdet, et.
al.


Transmission electron microscopic study of early stage gamma(1)
(Ag(2)Hg(3)) and beta(1) (Ag-Hg) phases: technical note.Incomplete
reaction, residual free mercury, and high volatility of mercury are
often major causes of the discrepancy in chemical composition of
gamma(1) measured by different methods.
from Dent Mater - Jul 2002 - J H Chern Lin, K I Chen


Occupational hygiene practices of dentists in southern
Thailand.OBJECTIVES: The aims of this study were to investigate the
prevalence and nature of infection control, radiation control and
handling of mercury, reported by dentists in southern Thailand.
from Int Dent J - Feb 2001 - P A Leggat, S Chowanadisai, B
Kukiattrakoon, et. al.


New approaches in the development of DNA sensors: hybridization and
electrochemical detection of DNA and RNA at two different surfaces.Up
to now, the development of the electrochemical DNA hybridization
sensors relied on solid electrodes, on which both the hybridization
and detection steps have been performed.
from Bioelectrochemistry - May 2002 - E Palecek, M Fojta


Determination of mercury in blood, urine and saliva for the biological
monitoring of an exposure from amalgam fillings in a group with
self-reported adverse health effects.It has been argued that the
release of mercury from amalgam fillings is of toxicological
relevance.
from Int J Hyg Environ Health - Apr 2002 - Holger Zimmer, Heidi
Ludwig, Michael Bader, et. al.


Urinary mercury excretion following amalgam filling in
children.OBJECTIVES: Dental amalgam is the major source of inorganic
mercury exposure in the general population.
from J Toxicol Clin Toxicol - Jan 2001 - M Khordi-Mood, A R
Sarraf-Shirazi


Placental transfer of mercury in pregnant rats which received dental
amalgam restorations.Mercury vapor released from one, two and four
amalgam restorations in pregnant rats and mercury concentrations in
maternal and fetal organs were studied.
from Toxicology - Mar 2003 - Yoshifumi Takahashi, Shozo Tsuruta,
Michitoshi Arimoto, et. al.


Oral lichen planus and allergy to dental amalgam
restorations.OBJECTIVES: To determine contact allergies in patients
with oral lichen planus and to monitor the effect of partial or
complete replacement of amalgam fillings following a positive patch
test reaction to ammoniated mercury, metallic mercury, or amalgam.
from Arch Dermatol - Dec 2004 - Ronald Laeijendecker, Sybren K
Dekker, Piet M Burger, et. al.


[The dynamic change of mercury concentration in urine after amalgam
filled]OBJECTIVE: Through the regular examination of mercury
concentration in urine this study was to observe the dynamic change of
the mercury concentration in the patients with amalgam fillings so as
to find out the effect of amalgam on health.
from Shanghai Kou Qiang Yi Xue - Mar 2001 - J Wang


[Amalgam risk assessment with coverage of references up to
2005]Amalgam, which has been in use in dentistry for 150 years,
consists of 50 % elemental mercury and a mixture of silver, tin,
copper and zinc.
from Gesundheitswesen - Mar 2005 - J Mutter, J Naumann, H Walach

Disbarred Markey-- knows what Jason wants to know...........................

And. What Jason read.......................

Sure glad Mark is not responding to Jason.

UDP, and all of that.

"Mark Probert" wrote in message
...
JohnDoe wrote:
Jason Johnson wrote:
In article , Mark Probert
wrote:

http://www.pubmedcentral.nih.gov/art...?artid=1280342
Abstract
Thimerosal is a preservative that has been used in manufacturing
vaccines since the 1930s. Reports have indicated that infants can
receive ethylmercury (in the form of thimerosal) at or above the U.S.
Environmental Protection Agency guidelines for methylmercury exposure,
depending on the exact vaccinations, schedule, and size of the infant.
In this study we compared the systemic disposition and brain
distribution of total and inorganic mercury in infant monkeys after
thimerosal exposure with those exposed to MeHg. Monkeys were exposed to
MeHg (via oral gavage) or vaccines containing thimerosal (via
intramuscular injection) at birth and 1, 2, and 3 weeks of age. Total
blood Hg levels were determined 2, 4, and 7 days after each exposure.
Total and inorganic brain Hg levels were assessed 2, 4, 7, or 28 days
after the last exposure. The initial and terminal half-life of Hg in
blood after thimerosal exposure was 2.1 and 8.6 days, respectively,
which are significantly shorter than the elimination half-life of Hg
after MeHg exposure at 21.5 days. Brain concentrations of total Hg were
significantly lower by approximately 3-fold for the thimerosal-exposed
monkeys when compared with the MeHg infants, whereas the average
brain-to-blood concentration ratio was slightly higher for the
thimerosal-exposed monkeys (3.5 ± 0.5 vs. 2.5 ± 0.3). A higher
percentage of the total Hg in the brain was in the form of inorganic Hg
for the thimerosal-exposed monkeys (34% vs. 7%). The results indicate
that MeHg is not a suitable reference for risk assessment from exposure
to thimerosal-derived Hg. Knowledge of the toxicokinetics and
developmental toxicity of thimerosal is needed to afford a meaningful
assessment of the developmental effects of thimerosal-containing
vaccines.
--------------------
Clearly, the claim by the Mercury Militia that it accumulates after
each vaccination is not supported by this research. Ethyl Mercury, the
byproduct of thimerosal metabolism is eliminated rapidly, and is gone
before the next vaccination.

~~~~~~~~~~~~~~~~~~~~~~~~~~

How was this study funded?
~~~~~~~~~~~~~~~~~~~~~~~~~~

With money?

Jason really wants to know if he can trash the study as being funded by
BigPharma. He did not bother to read the full study which answers the
question he meant to ask: Who funded it?

Markey's buzz word. Whine.

Oh, wait. Now is it *obviously* Two minutes ago.
It was *He did not bother*.

Keep it up, Markey. Show your true colors.

"Mark Probert" wrote in message
...
Jason Johnson wrote:
In article , Mark Probert
wrote:

http://www.pubmedcentral.nih.gov/art...?artid=1280342
Abstract
Thimerosal is a preservative that has been used in manufacturing
vaccines since the 1930s. Reports have indicated that infants can receive
ethylmercury (in the form of thimerosal) at or above the U.S.
Environmental Protection Agency guidelines for methylmercury exposure,
depending on the exact vaccinations, schedule, and size of the infant. In
this study we compared the systemic disposition and brain distribution of
total and inorganic mercury in infant monkeys after thimerosal exposure
with those exposed to MeHg. Monkeys were exposed to MeHg (via oral
gavage) or vaccines containing thimerosal (via intramuscular injection)
at birth and 1, 2, and 3 weeks of age. Total blood Hg levels were
determined 2, 4, and 7 days after each exposure. Total and inorganic
brain Hg levels were assessed 2, 4, 7, or 28 days after the last
exposure. The initial and terminal half-life of Hg in blood after
thimerosal exposure was 2.1 and 8.6 days, respectively, which are
significantly shorter than the elimination half-life of Hg after MeHg
exposure at 21.5 days. Brain concentrations of total Hg were
significantly lower by approximately 3-fold for the thimerosal-exposed
monkeys when compared with the MeHg infants, whereas the average
brain-to-blood concentration ratio was slightly higher for the
thimerosal-exposed monkeys (3.5 ± 0.5 vs. 2.5 ± 0.3). A higher percentage
of the total Hg in the brain was in the form of inorganic Hg for the
thimerosal-exposed monkeys (34% vs. 7%). The results indicate that MeHg
is not a suitable reference for risk assessment from exposure to
thimerosal-derived Hg. Knowledge of the toxicokinetics and developmental
toxicity of thimerosal is needed to afford a meaningful assessment of the
developmental effects of thimerosal-containing vaccines.
--------------------
Clearly, the claim by the Mercury Militia that it accumulates after each
vaccination is not supported by this research. Ethyl Mercury, the
byproduct of thimerosal metabolism is eliminated rapidly, and is gone
before the next vaccination.

~~~~~~~~~~~~~~~~~~~~~~~~~~

How was this study funded?

Is that the best you can do? Whine about funding. Obviously, you did not
bother to even attempt to read it. You answer is at the link I posted. Do
your own homework. Read the study and try to find fault with methodology,
etc.

Sure glad Mark is not responding to Jason............................

"Mark Probert" wrote in message
...
Jason Johnson wrote:
In article , Mark Probert
wrote:

http://www.pubmedcentral.nih.gov/art...?artid=1280342
Abstract
Thimerosal is a preservative that has been used in manufacturing
vaccines since the 1930s. Reports have indicated that infants can receive
ethylmercury (in the form of thimerosal) at or above the U.S.
Environmental Protection Agency guidelines for methylmercury exposure,
depending on the exact vaccinations, schedule, and size of the infant. In
this study we compared the systemic disposition and brain distribution of
total and inorganic mercury in infant monkeys after thimerosal exposure
with those exposed to MeHg. Monkeys were exposed to MeHg (via oral
gavage) or vaccines containing thimerosal (via intramuscular injection)
at birth and 1, 2, and 3 weeks of age. Total blood Hg levels were
determined 2, 4, and 7 days after each exposure. Total and inorganic
brain Hg levels were assessed 2, 4, 7, or 28 days after the last
exposure. The initial and terminal half-life of Hg in blood after
thimerosal exposure was 2.1 and 8.6 days, respectively, which are
significantly shorter than the elimination half-life of Hg after MeHg
exposure at 21.5 days. Brain concentrations of total Hg were
significantly lower by approximately 3-fold for the thimerosal-exposed
monkeys when compared with the MeHg infants, whereas the average
brain-to-blood concentration ratio was slightly higher for the
thimerosal-exposed monkeys (3.5 ± 0.5 vs. 2.5 ± 0.3). A higher percentage
of the total Hg in the brain was in the form of inorganic Hg for the
thimerosal-exposed monkeys (34% vs. 7%). The results indicate that MeHg
is not a suitable reference for risk assessment from exposure to
thimerosal-derived Hg. Knowledge of the toxicokinetics and developmental
toxicity of thimerosal is needed to afford a meaningful assessment of the
developmental effects of thimerosal-containing vaccines.
--------------------
Clearly, the claim by the Mercury Militia that it accumulates after each
vaccination is not supported by this research. Ethyl Mercury, the
byproduct of thimerosal metabolism is eliminated rapidly, and is gone
before the next vaccination.

~~~~~~~~~~~~~~~~~~~~~~~~~~

How was this study funded?

Is that the best you can do? Whine about funding. Obviously, you did not
bother to even attempt to read it. You answer is at the link I posted. Do
your own homework. Read the study and try to find fault with methodology,
etc.

Welcome back, Max.

"Max C." wrote in message
oups.com...
Mark Probert wrote:
http://www.pubmedcentral.nih.gov/art...?artid=1280342

Abstract
Thimerosal is a preservative that has been used in manufacturing
vaccines since the 1930s. Reports have indicated that infants can
receive ethylmercury (in the form of thimerosal) at or above the U.S.
Environmental Protection Agency guidelines for methylmercury exposure,
depending on the exact vaccinations, schedule, and size of the infant.
In this study we compared the systemic disposition and brain
distribution of total and inorganic mercury in infant monkeys after
thimerosal exposure with those exposed to MeHg. Monkeys were exposed to
MeHg (via oral gavage) or vaccines containing thimerosal (via
intramuscular injection) at birth and 1, 2, and 3 weeks of age. Total
blood Hg levels were determined 2, 4, and 7 days after each exposure.
Total and inorganic brain Hg levels were assessed 2, 4, 7, or 28 days
after the last exposure. The initial and terminal half-life of Hg in
blood after thimerosal exposure was 2.1 and 8.6 days, respectively,
which are significantly shorter than the elimination half-life of Hg
after MeHg exposure at 21.5 days. Brain concentrations of total Hg were
significantly lower by approximately 3-fold for the thimerosal-exposed
monkeys when compared with the MeHg infants, whereas the average
brain-to-blood concentration ratio was slightly higher for the
thimerosal-exposed monkeys (3.5 ± 0.5 vs. 2.5 ± 0.3). A higher
percentage of the total Hg in the brain was in the form of inorganic Hg
for the thimerosal-exposed monkeys (34% vs. 7%). The results indicate
that MeHg is not a suitable reference for risk assessment from exposure
to thimerosal-derived Hg. Knowledge of the toxicokinetics and
developmental toxicity of thimerosal is needed to afford a meaningful
assessment of the developmental effects of thimerosal-containing vaccines.

It gives me a sense of stability to know that I can go away for a few
months and come back to the same old junk that was being posted when I
left. It's unclear to me how the pro-vaccine group could use this
information to their advantage. Here's what I see when I read the
above info.

Brain concentrations of total Hg were
significantly lower by approximately 3-fold for the thimerosal-exposed
monkeys when compared with the MeHg infants, whereas the average
brain-to-blood concentration ratio was slightly higher for the
thimerosal-exposed monkeys (3.5 ± 0.5 vs. 2.5 ± 0.3).

OK, so they were lower. How would those infants otherwise be exposed
to MeHg in the real world? The point is that this comparison is
ridiculous. The fact that brain concentrations were lower in the
thimerosal group means nothing. They're STILL HIGHER if you compare
them to a group that received no mercury at all. Why in the world
would this stuy compare a thimerosal group with a MeHg group when their
shouldn't be any MeHg groups in the real world? Shouldn't the point of
the study be to compare real world scenarios?

A higher
percentage of the total Hg in the brain was in the form of inorganic Hg
for the thimerosal-exposed monkeys (34% vs. 7%).

hhhmmmm... there's that mercury in the brain again. Funny how that
paragraph mentions the half life of mercury in the blood but neglects
to mention it in the brain. Could that be because the brain has a much
harder time getting rid of the mercury than the blood? Also, shouldn't
the fact that the mercury in the brains of the thimerosal group was
mostly inorganic be a concern? I thought you pro-vaccine guys were
constantly saying that the end Hg product from thimerosal was all
organic.

The results indicate
that MeHg is not a suitable reference for risk assessment from exposure
to thimerosal-derived Hg. Knowledge of the toxicokinetics and
developmental toxicity of thimerosal is needed to afford a meaningful
assessment of the developmental effects of thimerosal-containing vaccines.

So, basically, this last piece is saying exactly what it *should*
say... this study is meaningless, since more study is needed for "a
meaningful assessment." Leave it to the pro-vaccine side to disagree
with that and try to use it to prove some sort of point.

You should pick apart your studies a little better before posting them.

Max.

"john" wrote:


"Peter Bowditch" wrote in message
.. .

You consistently state the same bull**** --that autism couldn't be caused
by more than one thing


Well, which is it? In each case, Mercola says that it is THE cause. Is
he lying? What else does he lie about?

And I have never said that autism can't be caused by more than one
thing. It could be caused by more than one gene, for example. It's
just not caused by one of or any combination of fluoridation,
vaccination or pasteurisation.
--

So, as long as it's not caused by your favourites--fluoride, mercury,
vaccines or heated milk.


As I know it isn't, what have I to worry about?
--
Peter Bowditch aa #2243
The Millenium Project http://www.ratbags.com/rsoles
Australian Council Against Health Fraud http://www.acahf.org.au
Australian Skeptics http://www.skeptics.com.au
To email me use my first name only at ratbags.com

In article , Mark Probert
wrote:

Jason Johnson wrote:
In article , Mark Probert
wrote:

Jason Johnson wrote:
In article , Mark Probert
wrote:

http://www.pubmedcentral.nih.gov/art...?artid=1280342

Abstract
Thimerosal is a preservative that has been used in manufacturing
vaccines since the 1930s. Reports have indicated that infants can
receive ethylmercury (in the form of thimerosal) at or above the U.S.
Environmental Protection Agency guidelines for methylmercury exposure,
depending on the exact vaccinations, schedule, and size of the infant.
In this study we compared the systemic disposition and brain
distribution of total and inorganic mercury in infant monkeys after
thimerosal exposure with those exposed to MeHg. Monkeys were exposed to
MeHg (via oral gavage) or vaccines containing thimerosal (via
intramuscular injection) at birth and 1, 2, and 3 weeks of age. Total
blood Hg levels were determined 2, 4, and 7 days after each exposure.
Total and inorganic brain Hg levels were assessed 2, 4, 7, or 28 days
after the last exposure. The initial and terminal half-life of Hg in
blood after thimerosal exposure was 2.1 and 8.6 days, respectively,
which are significantly shorter than the elimination half-life of Hg
after MeHg exposure at 21.5 days. Brain concentrations of total Hg were
significantly lower by approximately 3-fold for the thimerosal-exposed
monkeys when compared with the MeHg infants, whereas the average
brain-to-blood concentration ratio was slightly higher for the
thimerosal-exposed monkeys (3.5 ± 0.5 vs. 2.5 ± 0.3). A higher
percentage of the total Hg in the brain was in the form of inorganic Hg
for the thimerosal-exposed monkeys (34% vs. 7%). The results indicate
that MeHg is not a suitable reference for risk assessment from exposure
to thimerosal-derived Hg. Knowledge of the toxicokinetics and
developmental toxicity of thimerosal is needed to afford a meaningful
assessment of the developmental effects of thimerosal-containing
vaccines.

--------------------

Clearly, the claim by the Mercury Militia that it accumulates after each
vaccination is not supported by this research. Ethyl Mercury, the
byproduct of thimerosal metabolism is eliminated rapidly, and is gone
before the next vaccination.

~~~~~~~~~~~~~~~~~~~~~~~~~~

How was this study funded?

Is that the best you can do? Whine about funding. Obviously, you did not
bother to even attempt to read it. You answer is at the link I posted.
Do your own homework. Read the study and try to find fault with
methodology, etc.

~~~~~~~~~~~~~~~~~~~~~~~~~

Mark,
I read the study that is posted above and have read other research studies
that have had similar conclusions. I have also read other research studies
that have had different conclusions.

I posted the link since the entire study is available.

Now, specify what other *studies*, with references, have different
findings? I cannot find fault with this studies methodology.

Chemistry does not change.

~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

Mark,
I read various research studies but only saved one report which mentioned
various studies showing that thimerosal was the cause of autism. I seem to

recall that you read that report which I have posted in this newsgroup
several different times and you discounted every one of those studies.
Upon request, I will post it again since I saved it.
I read the study results that you posted above and I will NOT discount it
since I have NEVER done any research related to the genes of monkeys. It's
my opinion that children that develop autism have some defective genes.
Upon request, I can post a research study that mentions that subject.
I believe that the defective genes make it impossible for children to
process mercury which is the reason they develop autism when exposed to
mercury.
I don't know whether monkeys have those same defective genes.
I do believe that some of the studies that indicate that thimerosal does
NOT cause autism did NOT include any children in those studies that had
the defective genes. Only a small number of children have the defective
genes.
jason
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

"Jason Johnson" wrote in message
...
In article , Mark Probert
wrote:

Jason Johnson wrote:
In article , Mark Probert
wrote:

Jason Johnson wrote:
In article , Mark Probert
wrote:

http://www.pubmedcentral.nih.gov/art...?artid=1280342

Abstract
Thimerosal is a preservative that has been used in manufacturing
vaccines since the 1930s. Reports have indicated that infants can
receive ethylmercury (in the form of thimerosal) at or above the
U.S.
Environmental Protection Agency guidelines for methylmercury
exposure,
depending on the exact vaccinations, schedule, and size of the
infant.
In this study we compared the systemic disposition and brain
distribution of total and inorganic mercury in infant monkeys after
thimerosal exposure with those exposed to MeHg. Monkeys were exposed
to
MeHg (via oral gavage) or vaccines containing thimerosal (via
intramuscular injection) at birth and 1, 2, and 3 weeks of age.
Total
blood Hg levels were determined 2, 4, and 7 days after each
exposure.
Total and inorganic brain Hg levels were assessed 2, 4, 7, or 28
days
after the last exposure. The initial and terminal half-life of Hg in
blood after thimerosal exposure was 2.1 and 8.6 days, respectively,
which are significantly shorter than the elimination half-life of Hg
after MeHg exposure at 21.5 days. Brain concentrations of total Hg
were
significantly lower by approximately 3-fold for the
thimerosal-exposed
monkeys when compared with the MeHg infants, whereas the average
brain-to-blood concentration ratio was slightly higher for the
thimerosal-exposed monkeys (3.5 ± 0.5 vs. 2.5 ± 0.3). A higher
percentage of the total Hg in the brain was in the form of inorganic
Hg
for the thimerosal-exposed monkeys (34% vs. 7%). The results
indicate
that MeHg is not a suitable reference for risk assessment from
exposure
to thimerosal-derived Hg. Knowledge of the toxicokinetics and
developmental toxicity of thimerosal is needed to afford a
meaningful
assessment of the developmental effects of thimerosal-containing
vaccines.

--------------------

Clearly, the claim by the Mercury Militia that it accumulates after
each
vaccination is not supported by this research. Ethyl Mercury, the
byproduct of thimerosal metabolism is eliminated rapidly, and is
gone
before the next vaccination.

~~~~~~~~~~~~~~~~~~~~~~~~~~

How was this study funded?

Is that the best you can do? Whine about funding. Obviously, you did
not
bother to even attempt to read it. You answer is at the link I posted.
Do your own homework. Read the study and try to find fault with
methodology, etc.

~~~~~~~~~~~~~~~~~~~~~~~~~

Mark,
I read the study that is posted above and have read other research
studies
that have had similar conclusions. I have also read other research
studies
that have had different conclusions.

I posted the link since the entire study is available.

Now, specify what other *studies*, with references, have different
findings? I cannot find fault with this studies methodology.

Chemistry does not change.

Completely independant of taking any side here, Chemistry (the observation
of elemental constructs and reactions) always changes.



~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

Mark,
I read various research studies but only saved one report which mentioned
various studies showing that thimerosal was the cause of autism. I seem to

recall that you read that report which I have posted in this newsgroup
several different times and you discounted every one of those studies.
Upon request, I will post it again since I saved it.
I read the study results that you posted above and I will NOT discount it
since I have NEVER done any research related to the genes of monkeys. It's
my opinion that children that develop autism have some defective genes.
Upon request, I can post a research study that mentions that subject.
I believe that the defective genes make it impossible for children to
process mercury which is the reason they develop autism when exposed to
mercury.
I don't know whether monkeys have those same defective genes.
I do believe that some of the studies that indicate that thimerosal does
NOT cause autism did NOT include any children in those studies that had
the defective genes. Only a small number of children have the defective
genes.
jason
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

In article , "Vernon"
there@atthere wrote:

"Jason Johnson" wrote in message
...
In article , Mark Probert
wrote:

Jason Johnson wrote:
In article , Mark Probert
wrote:

Jason Johnson wrote:
In article , Mark Probert
wrote:

http://www.pubmedcentral.nih.gov/art...?artid=1280342

Abstract
Thimerosal is a preservative that has been used in manufacturing
vaccines since the 1930s. Reports have indicated that infants can
receive ethylmercury (in the form of thimerosal) at or above the
U.S.
Environmental Protection Agency guidelines for methylmercury
exposure,
depending on the exact vaccinations, schedule, and size of the
infant.
In this study we compared the systemic disposition and brain
distribution of total and inorganic mercury in infant monkeys after
thimerosal exposure with those exposed to MeHg. Monkeys were exposed
to
MeHg (via oral gavage) or vaccines containing thimerosal (via
intramuscular injection) at birth and 1, 2, and 3 weeks of age.
Total
blood Hg levels were determined 2, 4, and 7 days after each
exposure.
Total and inorganic brain Hg levels were assessed 2, 4, 7, or 28
days
after the last exposure. The initial and terminal half-life of Hg in
blood after thimerosal exposure was 2.1 and 8.6 days, respectively,
which are significantly shorter than the elimination half-life of Hg
after MeHg exposure at 21.5 days. Brain concentrations of total Hg
were
significantly lower by approximately 3-fold for the
thimerosal-exposed
monkeys when compared with the MeHg infants, whereas the average
brain-to-blood concentration ratio was slightly higher for the
thimerosal-exposed monkeys (3.5 ± 0.5 vs. 2.5 ± 0.3). A higher
percentage of the total Hg in the brain was in the form of inorganic
Hg
for the thimerosal-exposed monkeys (34% vs. 7%). The results
indicate
that MeHg is not a suitable reference for risk assessment from
exposure
to thimerosal-derived Hg. Knowledge of the toxicokinetics and
developmental toxicity of thimerosal is needed to afford a
meaningful
assessment of the developmental effects of thimerosal-containing
vaccines.

--------------------

Clearly, the claim by the Mercury Militia that it accumulates after
each
vaccination is not supported by this research. Ethyl Mercury, the
byproduct of thimerosal metabolism is eliminated rapidly, and is
gone
before the next vaccination.

~~~~~~~~~~~~~~~~~~~~~~~~~~

How was this study funded?

Is that the best you can do? Whine about funding. Obviously, you did
not
bother to even attempt to read it. You answer is at the link I posted.
Do your own homework. Read the study and try to find fault with
methodology, etc.

~~~~~~~~~~~~~~~~~~~~~~~~~

Mark,
I read the study that is posted above and have read other research
studies
that have had similar conclusions. I have also read other research
studies
that have had different conclusions.

I posted the link since the entire study is available.

Now, specify what other *studies*, with references, have different
findings? I cannot find fault with this studies methodology.

Chemistry does not change.

Completely independant of taking any side here, Chemistry (the observation
of elemental constructs and reactions) always changes.


~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

Vernon,
It depends on what Mark meant when he used the term "chemistry". Mark
should explain what the meant. Perhaps he was referring to "natural laws".

It's been over 25 years since I have taken any science classes but seem to
recall learning that natural laws never change.
Jason
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~